Growth Inhibition by Metabolites of γ-Hexachlorocyclohexane in Sphingobium japonicum UT26
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概要
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The growth of a γ-hexachlorocyclohexane (γ-HCH)-degrading bacterium Sphingobium japonicum (formerly Sphingomonas paucimobilis) UT26 in rich medium was inhibited by γ-HCH. This growth inhibition was not observed in a mutant that lacked the initial or second step enzymatic activity for γ-HCH degradation, suggesting that metabolites of γ-HCH are toxic to UT26. Two metabolites of γ-HCH, 2,5-dichlorophenol (2,5-DCP) and 2,5-dichlorohydroquinone (2,5-DCHQ), showed a direct toxic effect on UT26 and other sphingomonad strains. Because only 2,5-DCP accumulated during γ-HCH degradation, 2,5-DCP is thought to be a main compound for growth inhibition.
- 2006-04-23
著者
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Tsuda Masataka
Department Of Environmental Life Sciences Graduate School Of Life Sciences Tohoku University
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Tsuda Masataka
Department Of Environmental Life Science Graduate School Of Life Science Tohoku University
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NAGATA Yuji
Department of Biotechnology, University of Tokyo
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Ohtsubo Yoshiyuki
Department of Biotechnology, The University of Tokyo
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Ohtsubo Yoshiyuki
Department Of Environmental Life Sciences Graduate School Of Life Sciences Tohoku University
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Ohtsubo Yoshiyuki
Department Of Biotechnology The University Of Tokyo
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Nagata Yuji
Department Of Environmental Life Sciences Graduate School Of Life Sciences Tohoku University
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Nagata Yuji
Department Of Applied And Environmental Chemistry Kitami Institute Of Technology
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ENDO Ryo
Department of Environmental Life Sciences, Graduate School of Life Sciences, Tohoku University
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Endo Ryo
Department Of Environmental Life Sciences Graduate School Of Life Sciences Tohoku University
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