Hormonal Regulation of Cardiac Cystic Fibrosis Transmembrane Conductance Regulator Chloride Channels
スポンサーリンク
概要
- 論文の詳細を見る
The chloride conductance that arises from the stimulation of β-adrenoceptors has been shown to be carried by a cardiac isoform of cystic fibrosis transmembrane conductance regulator (CFTR) Cl- channels. This brief review will focus first upon the cellular signal transduction system for the activation of this type of Cl- channels and then its regulation by catecholamines, muscarinic agonists, endothelin-1 and angiotensin-II. Both in physiological and pathological conditions, the complex interaction of these agonists modulates the Cl- conductance, which is potentially arrhythmogenie by shortening the action potential duration and inducing the depolarization.
- International Heart Journal刊行会の論文
著者
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Horie Minoru
Division of Cardiology, kumamoto University Medical School, and Division of Cardiology, Shizuoka Cit
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Sasayama Shigetake
Division of Medicine, Hamamatsu Rosai Hospital, Shizuoka
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Horie Minoru
Department Of Cardiovascular Medicine Kyoto University Graduate School Of Medicine
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Sasayama Shigetake
Division Of Medicine Hamamatsu Rosai Hospital Shizuoka
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OBAYASHI Kazuhiko
Division of Cardiac Electrophysiology, the Third Department of Internal Medicine, Faculty of Medicin
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XIE Lai-Hua
Division of Cardiac Electrophysiology, the Third Department of Internal Medicine, Faculty of Medicin
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JAMES Andrew
Cell Physiology Group, Department of Medicine, Kings College London School of Medicine and Dentistry
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Obayashi Kazuhiko
Department Of Cardiovascular Medicine Kyoto University Graduate School Of Medicine
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XIE Lai-Hua
Division of Cardiac Electrophysiology, the Third Department of Internal Medicine, Faculty of Medicine, Kyoto University
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SASAYAMA Shigetake
Division of Cardiac Electrophysiology, the Third Department of Internal Medicine, Faculty of Medicine, Kyoto University
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HORIE Minoru
Division of Cardiac Electrophysiology, the Third Department of Internal Medicine, Faculty of Medicine, Kyoto University
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OBAYASHI Kazuhiko
Division of Cardiac Electrophysiology, the Third Department of Internal Medicine, Faculty of Medicine, Kyoto University
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JAMES Andrew
Cell Physiology Group, Department of Medicine, King's College London School of Medicine and Dentistry
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