Alterations in the Intrarenal Hemodynamics Following Changes in Ureteral Pressure in the Rabbit Kidney

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概要

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• Alterations in intrarenal hemodynamics following elevations of ureteral pressure were studied in the intact innervated and isolated perfused kidneys of rabbits.In the intact kidney increased ureteral pressure resulted in an increase in tissue pressure. Renal venous outflow showed at first a transient fall and then increased to or above the control value. A significant increase of tissue pressure over 50 mm.Hg resulted in a decrease in renal venous outflow in some cases. Total renal vascular resistance did not significantly change. Prevenous resistance decreased as ureteral pressure increased, while venous resistance increased. The medullary transit time prolonged following ureteral pressure elevations. Papaverine failed to increase renal venous outflow during ureteral occlusion. The contralateral ureteral blockade did not affect renal venous outflow in the experimental kidney.The effect of increased ureteral pressure on segmental vascular resistances in the isolated perfused kidney was qualitatively similar to those in the intact kidney.These findings suggest that the tissue pressure mechanism as well as the myogenic or active vasotonic mechanism participates in the intrarenal hemodynamic alterations following ureteral pressure elevation.

• International Heart Journal刊行会の論文

著者

• Yoshitomi Yawara

  1st Dept.of Intern.med. Facul. Of Univ. Of Tokyo

• YOSHITOSHI YAWARA

  First Dept. of Intern. Med., Facul. of Med., Univ. of Tokyo

• SEKI Kiyoshi

  First Department of Internal Medicine, Nippon Medical School

• AIZAWA Chikara

  First Department of Internal Medicine, Faculty of Medicine, University of Tokyo

• HONDA Nishio

  First Department of Internal Medicine, Faculty of Medicine, University of Tokyo

• MORIKAWA Akio

  First Department of Internal Medicine, Faculty of Medicine, University of Tokyo

• YOSHITOSHI Yawara

  First Department of Internal Medicine, Faculty of Medicine, University of Tokyo

• SEKI Kiyoshi

  First Department of Internal Medicine, Faculty of Medicine, University of Tokyo

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