フェナジン-5N-モノオキシドのイネ白葉枯病に対する作用機構

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1. The action mechanism of phenazine-5N一monooxide (II) against rice leaf b1ight bacterium was investigated relating to the electron transport system of the bacterium on the basis of the action mechanism of phenazine (I) already reported. 2. (II) was reduced non-biologica11y to (I) by the addition of L-ascorbate or L-cysteine or reduced bio1ogica11y in the presence of the bacterial ce11 suspension under anaerobic condition. The action of (II) against the bacterium was attributed to (I) formed after the reductive cleavage of the N-oxide bond. 3. (I) and (II) did not inhibit the succinate dehydrogenase according to the experimental evidences as for the succinate oxidase system and succinate-phenazine methosulfate reductase of the bacterium. They also did not inhibit the triosephosphate dehydrogenase. Various reducing reagents (including L-ascorbate and L-cysteine) had not the reverse action against inhibitory action of (I) or (II). 4. In the presence of (I) or (II) and succinate as the substrate, the reduction of cytochrome b 1 in the succinate oxidase system of the bacterium was sti11 observed, however the reduction of cytochromec and a l were suppressed. The apperent respiration observed by manometric or po1arographic experiments under the presence of (I) or (II) and succinate as the substrate suggested the operation of the by-pass electron flow from succinate dehydrogenase or cytochrome b l to atomospheric oxygen through the redox system; (I) ⇄ dihydrophenazine. 5. In the presence of (I) or (II) under a 1imited incubation condition, inhibition against the uptake of ortho-phosphate and the dissimilation of glucose was observed with the s1ight acceleration of the apparent respiration.
日本植物病理学会の論文
1969-06-30

フェナジン-5N-モノオキシドのイネ白葉枯病に対する作用機構

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