タバコ・モザイク・ウイルスに全身感染した葉の紫外線照射による壊死斑の誘導

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It was reported that leaves from hosts which normally become systemically infected with tobacco mosaic virus (TMV) developed necrotic lesions if they were treated with actinomycin D. The present report describes that ultraviolet light (UV) irradiation induced necrotic lesions on both systemic and local lesion hosts systemically infected with TMV. When leaves of N. glutinosa, Xanthi-nc, or Samsun NN tobacco, inoculated with TMV and incubated at 30C under continuous illumination of 6,000 lux, were irradiated with UV on the upper leaf surface and incubated at light intensities below 200 lux, necrotic lesions appeared on the treated leaves. However, no lesions appeared on the treated leaves when UV irradiated leaves were incubated at 6,000 lux. Similar necrotic lesions appeared on TMV-infected leaves of the systemic hosts, Xanthi or Samsun tobacco, by UV irradiation. It was also found that water-soaked lesions developed on the leaves of both kinds of hosts systemically infected with TMV when they were incubated at 30C for more than 10 hr in complete darkness, even if infected leaves were not irradiated with UV. It was apparent that either of treatments of UV irradiation, dark incubation, and actinomycin D which were able to induce necrotic lesions on the leaves systemically infected with TMV, inhibited nucleic acid and protein syntheses more effectively in the cytoplasmic ribosomal fraction of the infected leaves than that in TMV fraction. In the case of UV irradiation followed by incubation at 6,000 lux, and of treatments of formycin B, blasticidin S, and rifampicin all of which were not able to induce necrotic lesions, nucleic acid and protein syntheses were inhibited at the same rate in both TMV and ribosomal fractions. The nature of induction of necrotic lesions on leaves systemically infected with TMV by the various treatments is discussed.
日本植物病理学会の論文
1979-04-25

タバコ・モザイク・ウイルスに全身感染した葉の紫外線照射による壊死斑の誘導

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