Kaneko Masayuki | Department Of Pharmacology Faculty Of Pharmaceutical Sciences Chiba Institute Of Science

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著作論文

A ubiquitin ligase HRD1 promotes the degradation of Pael receptor, a substrate of Parkin.
Novel Functions of Ubiquitin Ligase HRD1 With Transmembrane and Proline-Rich Domains
A different pathway in the endoplasmic reticulum stress-induced expression of human HRD1 and SEL1 genes
Manganese Superoxide Dismutase Is Induced by Endoplasmic Reticulum Stress through IRE1-Mediated Nuclear Factor (NF)-κB and AP-1 Activation(Biochemistry/Molecular Biology)
Activation Signal of Nuclear Factor-κB in Response to Endoplasmic Reticulum Stress is Transduced via IRE1 and Tumor Necrosis Factor Receptor-Associated Factor 2
Expression of the Ubiquitin Ligase HRD1 in Neural Stem/Progenitor Cells of the Adult Mouse Brain
Activation of Ceramidase and Ceramide Kinase by Vanadate via a Tyrosine Kinase-Mediated Pathway
Possible Involvement of HRD1, a Novel Molecule Related to Endoplasmic Reticulum Stress, in Alzheimer's Disease (Forum Minireview : Molecular Approaches to the Treatment, Prophylaxis, and Diagnosis of Alzheimer's Disease)
Possible Involvement of Ubiquitin Ligase HRD1 Insolubilization in Amyloid β Generation
Correlation Between Decrease in Protein Levels of Ubiquitin Ligase HRD1 and Amyloid-β Production
Molecular Approaches to the Treatment, Prophylaxis, and Diagnosis of Alzheimer's Disease : Possible Involvement of HRD1, a Novel Molecule Related to Endoplasmic Reticulum Stress, in Alzheimer's Disease
Expression of the Ubiquitin Ligase HRD1 in Neural Stem/Progenitor Cells of the Adult Mouse Brain
Protective Effects of 4-Phenylbutyrate Derivatives on the Neuronal Cell Death and Endoplasmic Reticulum Stress

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