Protective Effects of 4-Phenylbutyrate Derivatives on the Neuronal Cell Death and Endoplasmic Reticulum Stress
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- A ubiquitin ligase HRD1 promotes the degradation of Pael receptor, a substrate of Parkin.
- Novel Functions of Ubiquitin Ligase HRD1 With Transmembrane and Proline-Rich Domains
- A different pathway in the endoplasmic reticulum stress-induced expression of human HRD1 and SEL1 genes
- Manganese Superoxide Dismutase Is Induced by Endoplasmic Reticulum Stress through IRE1-Mediated Nuclear Factor (NF)-κB and AP-1 Activation(Biochemistry/Molecular Biology)
- Activation Signal of Nuclear Factor-κB in Response to Endoplasmic Reticulum Stress is Transduced via IRE1 and Tumor Necrosis Factor Receptor-Associated Factor 2
- Expression of the Ubiquitin Ligase HRD1 in Neural Stem/Progenitor Cells of the Adult Mouse Brain
- Activation of Ceramidase and Ceramide Kinase by Vanadate via a Tyrosine Kinase-Mediated Pathway
- Possible Involvement of HRD1, a Novel Molecule Related to Endoplasmic Reticulum Stress, in Alzheimer's Disease (Forum Minireview : Molecular Approaches to the Treatment, Prophylaxis, and Diagnosis of Alzheimer's Disease)
- Possible Involvement of Ubiquitin Ligase HRD1 Insolubilization in Amyloid β Generation
- Correlation Between Decrease in Protein Levels of Ubiquitin Ligase HRD1 and Amyloid-β Production
- Molecular Approaches to the Treatment, Prophylaxis, and Diagnosis of Alzheimer's Disease : Possible Involvement of HRD1, a Novel Molecule Related to Endoplasmic Reticulum Stress, in Alzheimer's Disease
- Expression of the Ubiquitin Ligase HRD1 in Neural Stem/Progenitor Cells of the Adult Mouse Brain
- Protective Effects of 4-Phenylbutyrate Derivatives on the Neuronal Cell Death and Endoplasmic Reticulum Stress