Okuma Y | Hokkaido Univ. Sapporo Jpn
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概要
関連著者
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Okuma Y
Hokkaido Univ. Sapporo Jpn
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OKUMA Yasunobu
Departmen of Pharmacology, Graduate School of Pharmaceutical Sciences, Hokkaido University
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Okuma Yasunobu
Department Of Pharmacology Faculty Of Pharmaceutical Sciences Chiba Institute Of Sciences
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野村 靖幸
Yokohama College Of Pharmacy
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Nomura Yasuyuki
Department Of Pharmacology Faculty Of Pharmaceutical Sciences Hokkaido University
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NOMURA Yasuyuki
Departmen of Pharmacology, Graduate School of Pharmaceutical Sciences, Hokkaido University
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Nomura Yasuyuki
Dep. Of Pharmacotherapeutics Yokohama Coll. Of Pharmacy Jpn
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野村 靖幸
北海道大学薬学部薬効学講座
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野村 靖幸
北海道大学・大学院薬学研究科医療薬学講座薬理学分野
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野村 靖幸
北大薬
著作論文
- Deficits in learning ability and aging of skin in both strains of senescence-accelerated mouse (SAM) P8 and P10 : neuropathological, neurochemical, histological and pharmacological analysis(Chemical & Pharmacological study)
- Depressive Behavior and Alterations in Receptors for Dopamine and 5-Hydroxytryptamine in the Brain of the Senescence Accelerated Mouse (SAM)-P10
- Melatonin Inhibits the Central Sympatho-adrenomedullary Outflow in Rats
- Centrally Applied Nitric Oxide Donors Inhibit Vagally Evoked Rat Gastric Acid Secretion: Involvement of Sympathetic Outflow
- Correlation Between Decrease in Protein Levels of Ubiquitin Ligase HRD1 and Amyloid-β Production
- A ubiquitin ligase HRD1 promotes the degradation of Pael receptor, a substrate of Parkin.
- PI3K-Akt inactivation induced CHOP expression in endoplasmic reticulum-stressed cells
- Alteration of Leptin-Induced STAT3 Activation in the Brain of Senescence-Accelerated Mouse (SAM) P8(Pharmacology)
- Aspirin and Sodium Salicylate Protect Brain Cell Death Against Endoplasmic Reticulum Stress
- Possible Role of Interleukin-6 in PC12 Cell Death Induced by MPP^+ and Tetrahydroisoquinoline