Ikeda Yasuhiro | Division Of Cardiology Department Of Medicine And Clinical Science Yamaguchi University Graduate Sch

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著作論文

PJ-367 Ser136 Phosporylation of Bad by PKC Activation is a New Therapeutic Strategy for Improving the Muscle-derived Stem Cell Survival(PJ062,Regeneration (Basic, Clinical) (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese C
PJ-430 Domain2114-2149 within Ryanodine Receptor (RyR) Plays a Key Role for Stabilization of RyR2 in Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT)(PJ073,Arrhythmia, Others (Basic) 1 (A),Poster Session (Japanese),The 73rd Annual Scientific M
PJ-073 Protein Phosphatase 1 Beta is Most Significant Subunit in the Longitudinal Sarcoplasmic Reticulum and Controles Phospholamban Phosphorylation in Cardiomyocytes(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd
PJ-071 In Vivo Knockdown of Protein Phosphatase 1β Augments Cardiac Contracitility in Mice(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
PJ-068 Abnormally Tight Domain-Domain Interaction at Mutation Site could be a Primary Cause of Catecholaminergic Polymorphic Ventricular Tachycardia(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd Annual Scientific
5 Decreased affinity of calmodulin binding to RyR2 may cause leaky channel in CPVT-associated mutation(Young Investigator's Award for International Students Finalists Lecture (YIA International),The 73rd Annual Scientific Meeting of The Japanese Circulati
3 Correction of RyR2 Defects as a New Therapeutic Strategy against Heart Failure(Novel Therapeutic Strategies against Heart Failure,KSC-JCS Joint Symposium (KSC-JCS Joint Symposium) (I),The 73rd Annual Scientific Meeting of The Japanese Circulation Societ
Defective Ca^ Cycling as a Key Pathogenic Mechanism of Heart Failure(Molecular Mechanisms)
FRS-018 Marked Increase in Spontaneous Ca2+ Sparks In Response To β-stimulation As A Critical Cause Of Catecholaminergic Polymorphic Ventricular Tachycardia(New Insights into Basic Mechanism of Arrhythmia(A),Featured Research Session,The 72nd Annual Scien
FRS-017 Enhanced Sensitivity of the Cardiac Ryanodine Receptor to Activation by Luminal Ca^ as a Primary Cause of CPVT(New Insights into Basic Mechanism of Arrhythmia(A),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circ
4 Phospho-regulation of Sarcoplasmic Reticulum Microdomain in Heart Failure(Symposium 7 (SY-07) (M) Molecular Mechanism and Novel Therapeutic Target in Heart Failure,Special Program,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PJ-765 Scavenging Free Radicals by Low-dose Carvedilol Prevents Redox-dependent Ca^ Leak via Stabilization of Ryanodine Receptor in Heart Failure(Development and differentiation-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society
OJ-249 Interruption of Specific Domain-domain Interaction within Ryanodine Receptor Mediates Stabilization of Ryanodine Receptor in Failing Hearts(Heart failure, basic-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Tongue Muscle-Derived Stem Cells Express Connexin 43 and Improve Cardiac Remodeling and Survival After Myocardial Infarction in Mice
PE-164 Defective Inter-domain Interaction may Cause Spontaneous Ca^ Release via Reduced Affinity of Calmodulin Binding to RyR2 in Failing Hearts(PE028,Heart Failure, Basic (M),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese
Effects of In Vivo Gene Transfer of Fibroblast Growth Factor-2 on Cardiac Function and Collateral Vessel Formation in the Microembolized Rabbit Heart
OJ-249 Tenascin-C is Tissue Protective during the Development of Abdominal Aortic Aneurysm in Mice(OJ42,Peripheral Circulation/Vascular Disease (Pathophysiology, Basic) (H),Oral Presentation (Japanese),The 73rd Annual Scientific Meeting of The Japanese Ci
PE-190 Involvement of JNK2 in Mechanosensing and Proinflammatory Signaling during Development of Aortic Aneurysm(PE032,Peripheral Circulation/Vascular Disease (Pathophysiology, Basic) (H),Poster Session (English),The 73rd Annual Scientific Meeting of The
PE-179 JNK Activation Causes Endoplasmic Reticulum Stress Response in Adipocytes to Suppress The Adiponectin Secretion In Vivo(Diabetes/Obesity/Metabolic syndrome(05)(H),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulatio
PE-115 Critical Role of JNK2 for Development of Abdominal Aortic Aneurysm in Mice(Peripheral circulation/Vascular disease(03)(H),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OJ-092 Protein Phosphatase 1 β is the Major Phospholamban Phosphatase that Negatively Regulates Cardiac Contraction and Relaxation.(Heart failure, basic(02)(M),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Soci
2 Critical Role of JNK in Chronic Inflammation of Abdominal Aortic Aneurysms(Chronic Inflammation and Cardiovascular Diseases,Roundtable Discussion 7 (RT-07) (H),Special Program,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
Inhibitor-1 is Potential Target for Enhancing Sarcoplasmic Reticulum Ca^ Loading in Failing Hearts

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