Ohkusa Tomoko | Department Of Medical Bioregulation Division Of Cardiovascular Medicine Yamaguchi University School

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著作論文

OE-425 Changes of Adhesion Junctions Precede the Gap Junction Remodeling during the Development of Heart Failure(OE72,Arrhythmia, Mechanism (A),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
Effects of Aldosterone on Cx43 Gap Junction Expression in Neonatal Rat Cultured Cardiomyocytes
PJ-068 Abnormally Tight Domain-Domain Interaction at Mutation Site could be a Primary Cause of Catecholaminergic Polymorphic Ventricular Tachycardia(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd Annual Scientific
5 Decreased affinity of calmodulin binding to RyR2 may cause leaky channel in CPVT-associated mutation(Young Investigator's Award for International Students Finalists Lecture (YIA International),The 73rd Annual Scientific Meeting of The Japanese Circulati
PJ-765 Scavenging Free Radicals by Low-dose Carvedilol Prevents Redox-dependent Ca^ Leak via Stabilization of Ryanodine Receptor in Heart Failure(Development and differentiation-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society
PJ-103 Defective Domain-Domain Interaction within Ryanodine Receptor as a Critical Cause of Diastolic Ca^ Spark in Cardiomyocytes(Excitation-contraction coupling/Ion channel, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
AT_1 Receptor Antagonist Restores Cardiac Ryanodine Receptor Function, Rendering Isoproterenol-Induced Failing Heart Less Susceptible to Ca^-Leak Induced by Oxidative Stress(Experimental Investigation)
PJ-200 Aldosterone Modulated Gap Junction Remodeling through Mineralocorticoid Receptor(Arrhythmia, basic(05)(A),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
Beneficial Effects of the Dual L- and T-Type Ca^ Channel Blocker Efonidipine on Cardiomyopathic Hamsters
PE-132 Aldosterone Upregulates l_f and I_ Channels in Rat Ventricular Myocytes and Enhances Their Spontaneous Excitations(Arrhythmia, basic-3 (A) PE22,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulatio
Syncope Induced by Tobacco Smoking in the Head-up Position
PE-164 Defective Inter-domain Interaction may Cause Spontaneous Ca^ Release via Reduced Affinity of Calmodulin Binding to RyR2 in Failing Hearts(PE028,Heart Failure, Basic (M),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese
OJ-143 Urinary 8-Hydroxy-2' -Deoxyguanosine Level reflects symptomatic status and severity of systolic dysfunction in Patients with Chronic Heart Failure(Heart failure, clinical(05)(M),Oral Presentation (Japanese),The 72nd Annual Scientific Meeting of the
PJ-035 Aldosterone is an Important Arrhythmogenic Substrate by Modulating the Gap Junction Channel Protein Connexin43(Arrhythmia, basic-5, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PJ-034 T-type Ca^ Channel Modulates Gap Junction Remodeling through Renin-Angiotensin-Aldosterone System(Arrhythmia, basic-5, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
1 Gap Junction Remodeling is an Important Arrhythmogenic Substrate during the Development of Heart Failure(Heart Failure and Arrhythmia, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PE-129 Aldosterone Modulates Gap Junction Remodeling in Cultured Rat Ventricular Myocytes(Arrhythmia, basic-3 (A) PE22,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Defective Regulation of Inter-domain Interactions within the Ryanodine Receptor Plays a Key Role in the Pathogenesis of Heart Failure (Excitation-contraction Coupling/Ion Channel 2 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Societ
Overexpression of Sorcin Prevents Isoproterenol-Induced Apoptosis in Cultured Neonatal Rat Cardiomyocytes (Apoptosis/Necrosis/Regeneration 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Effects of Aldosterone on Expression of Gap Junction Protein Connexin43 in Cultured Neonatal Rat Ventricular Myocytes(Arrhythmia, Basic 2 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Gap Junction Remodeling is an Arrhythmogenic Substrate during the Development of Heart Failure in Cardiomyopathic Hamster(Arrhythmia, Basic 1 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Alterations in gap junction proteins in atrial myocardial tissue in patients with chronic atrial fibrillation
Altered expression of connexin43 contributes to the arrhythmogenic substrate during the development of heart failure in cardiomyopathic hamster
Differed Ca^ Regulatory Effect of Candesartan or a New Cardioprotective Agent JTV519 on Improvement of Isoproterenol-Induced Dysfunction in Rats
Valsartan Restores Normal FKBP12.6-RyR Interaction by a Strong Inhibition of PKA-Mediated Hyperphosphorylation of RyR in Heart Failure
Candesartan Prevents PKA-hyperphosphorylation, being Less Susceptible to Ca2+ Leak through Ryanodine Receptor in Isoproterenol-induced Failing Rats(Heart Failure, Basic 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OJ-016 Defective Regulation of Inter-Domain Interaction within Ryanodine Receptor as a Cause of Heart Failure(Excitation-Contraction Coupling/Ion Channel (A) : OJ3)(Oral Presentation (Japanese))
Stabilization of Calcium Release Channel (Ryanodine Receptor) Is Involved in the Cardioprotective Mechanism by Angiotensin II Receptor Blocker in Heart Failure
1 Structural Remodeling in Atrail Fibrillation as Arrhythmogenic Substrates : Tachyarrhythmia and Gap Junction Remodeling(Plenary Session 3 (PL3) (A) : Atrial Fibrillation : From bench to bedside)(Special Program)
High Ambient Pressure Produces Hypertrophy and Up-Regulates Cardiac Sarcoplasmic Reticulum Ca^ Regulatory Proteins in Cultured Rat Cardiomyocytes
A novel cardioprotective agent, JTV519 improves cardiac function by restoring Ca^ -release function of cardiac sarcoplasmic reticulum in heart failure
Propranolol prevents the development of heart failure by FKBP12.6-mediated stabilization of ryanodine receptor in sarcoplasmic reticulum
FKBP12.6-mediated stabilization of calcium release channel (ryanodine receptor) as a novel therapeutic target in heart failure
Functional and Structural Remodeling of Cardiomyocytes as Arrhythmogenic Substrates : Alterations in Ca^-Regulatory Proteins and Gap Junction Proteins in Atrial Fibrillation
Extrinsic rapid electrical stimulation modulates sarcoplasmic reticulum Ca^ proteins of cultured rat cardiomyocytes
OE-135 Gap junction remodeling as arrhythmogenic substrates during development heart failure in cardiomyopathic hamster hearts(Arrhythmia, Basic 4 (A) : OE16)(Oral Presentation (English))
Modulation of Cardiac Ryanodine Receptor by Sorcin in UM-X7.1 Cardiomyopathic Hamster Hearts
Rapid Electrical Stimulation of Contraction Modulates Gap-Junction Protein in Cultured Neonatal Rat Cardiomyocytes
Effects of Amiodarone on Gap-Junction Proteins in Cultured Rat Cardiomyocytes Exposed to Rapid Electrical Stimulation of Contraction
Effects of Efonidipine, a Novel Ca^ Channel Antagonist with T-Type Activity, on Cardiomyopathic Hamster Hearts during Development of Heart Failure
FRS-190 Sorcin modulates cardiac ryanodine receptors in cardiomyopathic hamster hearts(Heart Failure : Basic and Clinical Viewpoint (M) : FRS22)(Featured Research Session (English))
Conformational change of the foot protein of sarcoplasmic reticulum as an initial event of calcium release.

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