Doi Masahiro | Department Of Medicine And Clinical Science Yamaguchi University Graduate School Of Medicine

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概要

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著作論文

PJ-430 Domain2114-2149 within Ryanodine Receptor (RyR) Plays a Key Role for Stabilization of RyR2 in Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT)(PJ073,Arrhythmia, Others (Basic) 1 (A),Poster Session (Japanese),The 73rd Annual Scientific M
PJ-068 Abnormally Tight Domain-Domain Interaction at Mutation Site could be a Primary Cause of Catecholaminergic Polymorphic Ventricular Tachycardia(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd Annual Scientific
PJ-564 Reduction of In-stent Restenosis by Undersized Stenting : A Study with Quantitative Coronary Analysis(Restenosis after Angioplasty, Basic/Clinical 6 (IHD) : PJ94)(Poster Session (Japanese))
AT_1 Receptor Antagonist Restores Cardiac Ryanodine Receptor Function, Rendering Isoproterenol-Induced Failing Heart Less Susceptible to Ca^-Leak Induced by Oxidative Stress(Experimental Investigation)
Effects of Flecainide on the Electrophysiological Properties of Atrial Vulnerability in Humans
Factors Related to Threshold of Intra -Cardiac Atrial- Defibrillation : Excitable Period Obtained by Using Autocorrelogram
PE-164 Defective Inter-domain Interaction may Cause Spontaneous Ca^ Release via Reduced Affinity of Calmodulin Binding to RyR2 in Failing Hearts(PE028,Heart Failure, Basic (M),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese
Defective Regulation of Inter-domain Interactions within the Ryanodine Receptor Plays a Key Role in the Pathogenesis of Heart Failure (Excitation-contraction Coupling/Ion Channel 2 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Societ
Ventricular Conduction in Idiopathic Ventricular Fibrillation without Right Precordial ST Elevation : Comparative Study with Brugada Syndrome
Differed Ca^ Regulatory Effect of Candesartan or a New Cardioprotective Agent JTV519 on Improvement of Isoproterenol-Induced Dysfunction in Rats
Valsartan Restores Normal FKBP12.6-RyR Interaction by a Strong Inhibition of PKA-Mediated Hyperphosphorylation of RyR in Heart Failure
OJ-016 Defective Regulation of Inter-Domain Interaction within Ryanodine Receptor as a Cause of Heart Failure(Excitation-Contraction Coupling/Ion Channel (A) : OJ3)(Oral Presentation (Japanese))
Stabilization of Calcium Release Channel (Ryanodine Receptor) Is Involved in the Cardioprotective Mechanism by Angiotensin II Receptor Blocker in Heart Failure
DPE-015 The Effect of Bepridil on the Progression of Paroxysmal Atrial Fibrillation to Chronic Form Compared with Class I Anti-arrhythmic Agent(DPE03,Atrial/Supraventricular Arrhythmia (Clinical/Treatment) (A),Digital Poster Session (English),The 73rd Ann
A novel cardioprotective agent, JTV519 improves cardiac function by restoring Ca^ -release function of cardiac sarcoplasmic reticulum in heart failure
Propranolol prevents the development of heart failure by FKBP12.6-mediated stabilization of ryanodine receptor in sarcoplasmic reticulum
FKBP12.6-mediated stabilization of calcium release channel (ryanodine receptor) as a novel therapeutic target in heart failure

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