Kobayashi Shigeki | Division Of Cardiology Department Of Medicine And Clinical Science Yamaguchi University Graduate Sch

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著作論文

Products of Thymine Oxygenation by a Non-heme Oxygenation Model, FeII(MeCN)62+–Ac2O–H2O2, and the Transition State Model between Oxoiron and Thymine
PJ-573 Dantrolene Prevents Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT) by Correcting Abnormal Ca^ Release through the Ryanodine Receptor(PJ096,Arrhythmia, Others (Clinical/Pathophysiology) 4 (A),Poster Session (Japanese),The 73rd Annu
PJ-430 Domain2114-2149 within Ryanodine Receptor (RyR) Plays a Key Role for Stabilization of RyR2 in Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT)(PJ073,Arrhythmia, Others (Basic) 1 (A),Poster Session (Japanese),The 73rd Annual Scientific M
PJ-375 Carperitide, Atrial Natriuretic Peptide, Improves Hemoglobin Level in Acute Heart Failure(PJ063,Heart Failure (Treatment) 1 (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
PJ-068 Abnormally Tight Domain-Domain Interaction at Mutation Site could be a Primary Cause of Catecholaminergic Polymorphic Ventricular Tachycardia(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd Annual Scientific
5 Decreased affinity of calmodulin binding to RyR2 may cause leaky channel in CPVT-associated mutation(Young Investigator's Award for International Students Finalists Lecture (YIA International),The 73rd Annual Scientific Meeting of The Japanese Circulati
3 Correction of RyR2 Defects as a New Therapeutic Strategy against Heart Failure(Novel Therapeutic Strategies against Heart Failure,KSC-JCS Joint Symposium (KSC-JCS Joint Symposium) (I),The 73rd Annual Scientific Meeting of The Japanese Circulation Societ
Defective Ca^ Cycling as a Key Pathogenic Mechanism of Heart Failure(Molecular Mechanisms)
PJ-203 Dantrolene, a therapeutic agent for malignant hyperthermia, inhibits catecholaminergic polymorphic ventricular tachycardia : Insights from a RyR2 R2474S/+ knock-in mouse model(Arrhythmia, basic(05)(A),Poster Session(Japanese),The 72nd Annual Scient
FRS-018 Marked Increase in Spontaneous Ca2+ Sparks In Response To β-stimulation As A Critical Cause Of Catecholaminergic Polymorphic Ventricular Tachycardia(New Insights into Basic Mechanism of Arrhythmia(A),Featured Research Session,The 72nd Annual Scien
FRS-017 Enhanced Sensitivity of the Cardiac Ryanodine Receptor to Activation by Luminal Ca^ as a Primary Cause of CPVT(New Insights into Basic Mechanism of Arrhythmia(A),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circ
PJ-765 Scavenging Free Radicals by Low-dose Carvedilol Prevents Redox-dependent Ca^ Leak via Stabilization of Ryanodine Receptor in Heart Failure(Development and differentiation-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society
PJ-103 Defective Domain-Domain Interaction within Ryanodine Receptor as a Critical Cause of Diastolic Ca^ Spark in Cardiomyocytes(Excitation-contraction coupling/Ion channel, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
FRS-076 Correction of Defective Inter-Domain Interactions within Ryanodine Recepter by Dantrolene May be a Potent Therapeutic Strategy against Lethal Arrhythmia(Arrhythmia (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PJ-402 A Domain Which Has a Strong Effect to Stabilize the Ryanodine Receptor Exists inside the Ryanodine Receptor Itself(Heart failure, basic-5 (M) PJ68,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation
PJ-400 β-blocker Ameliorates Intracellular Ca^ Handling in Heart Failure via Correction of Defective Inter-domain Interaction within Ryanodine Receptor(Heart failure, basic-5 (M) PJ68,Poster Session (Japanese),The 70th Anniversary Annual Scientific Me
PJ-175 Defective Inter-domain Interaction within the Ryanodine Recepter Plays a Key Role in the Pathogenesis of Heart Failure(Excitation-contraction coupling/Ion channel-2 (A) PJ30,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting o
PE-164 Defective Inter-domain Interaction may Cause Spontaneous Ca^ Release via Reduced Affinity of Calmodulin Binding to RyR2 in Failing Hearts(PE028,Heart Failure, Basic (M),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese
OJ-143 Urinary 8-Hydroxy-2' -Deoxyguanosine Level reflects symptomatic status and severity of systolic dysfunction in Patients with Chronic Heart Failure(Heart failure, clinical(05)(M),Oral Presentation (Japanese),The 72nd Annual Scientific Meeting of the
OJ-243 A-type Natriuretic Peptide Improves Cardiac Function Partly through Suppression of Intracellular ROS in Heart Failure(OJ41,Heart Failure (Treatment) (M),Oral Presentation (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Soc
1 Defective Inter-domain Interaction within the Ryanodine Receptor Underlies a Common Pathogenic Mechanism of Heart Failure and Lethal Arrhythmia(Subcellular Remodeling in the Failing Heart,Symposium 10 (SY-10) (A),The 73rd Annual Scientific Meeting of Th
Differed Ca^ Regulatory Effect of Candesartan or a New Cardioprotective Agent JTV519 on Improvement of Isoproterenol-Induced Dysfunction in Rats
Valsartan Restores Normal FKBP12.6-RyR Interaction by a Strong Inhibition of PKA-Mediated Hyperphosphorylation of RyR in Heart Failure
OJ-019 Dantrolene Stabilizes Domain-Domain Interactions within the Ryanodine Receptor in Failing Hearts(Heart failure, basic-2 (M) OJ4,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Candesartan Prevents PKA-hyperphosphorylation, being Less Susceptible to Ca2+ Leak through Ryanodine Receptor in Isoproterenol-induced Failing Rats(Heart Failure, Basic 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
EFFECT OF AORTIC CHARACTERISTIC IMPEDANCE ON PRELOAD-AFTERLOAD MISMATCH IN CANINE HEART IN SITU
A novel cardioprotective agent, JTV519 improves cardiac function by restoring Ca^ -release function of cardiac sarcoplasmic reticulum in heart failure
Propranolol prevents the development of heart failure by FKBP12.6-mediated stabilization of ryanodine receptor in sarcoplasmic reticulum
FKBP12.6-mediated stabilization of calcium release channel (ryanodine receptor) as a novel therapeutic target in heart failure
CHARACTERIZATION OP SARCOPLASMIC RETICULUM FUNCTION IN HYPERTROPHIC RAT HEARTS INDUCED BY AORTIC COARCTATION
Different Systolic Loading Sequences Produced a Change of Remodeling in Hypertrophic Left Ventricle.
CHARACTERIZATION OF CALCIUM RELEASING CHANNEL OF SARCOPLASMIC RETICULUM IN HYPERTROPHIC RAT HEARTS INDUCED BY AORTIC COARCTATION
Diastereomer-Specific Effects of Double-Stranded Peptides Conjugated with-L-Tyr-L-Phe-or-L-Tyr-D-Phe-Residues on Tyrosine : Phosphorylation and Inhibition of src^NRK, A431, MCF-7, and DU145 Cell Growth
Extrinsic rapid electrical stimulation modulates sarcoplasmic reticulum Ca^ proteins of cultured rat cardiomyocytes
Potent 2,2-Diphenyl-1-picrylhydrazyl Radical-Scavenging Activity of Novel Antioxidants, Double-Stranded Tyrosine Residues Conjugating Pyrocatechol
Dantrolene, a Therapeutic Agent for Malignant Hyperthermia, Inhibits Catecholaminergic Polymorphic Ventricular Tachycardia in a RyR2^ Knock-In Mouse Model
Assessment of the Brain Areas Perfused by Superselective Intra-arterial Chemotherapy Using Single Photon Emission Computed Tomography with Technetium-99m-Hexamethyl-propyleneamine Oxime : Technical Note
-0108- CARDIAC SARCOPLASMIC RETICULUM CA^ RELEASE PLAYS AN IMPORTANT ROLE IN THE PROGRESSION OF CARDIAC HYPERTROPHY(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)
-0593- THE NUMBER OF CARDIAC RYANODINE RECEPTORS DECREASED IN VOLUME OVERLOAD CARDIAC HYPERTROPHY IN RATS.(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)

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