Ikeda Yasuhiro | Dept. Of Medical Bioregulation Division Of Cardioventricular Medicine Yamaguchi University School Of

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著作論文

PJ-367 Ser136 Phosporylation of Bad by PKC Activation is a New Therapeutic Strategy for Improving the Muscle-derived Stem Cell Survival(PJ062,Regeneration (Basic, Clinical) (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese C
Regression of abdominal aortic aneurysms by pharmacological inhibition of c-Jun N-terminal kinase
FRS-058 A Novel Stem Cell Source for Cardiomyocyte Regeneration(Regeneration Research in Cardiovascular Medicine (M) FRS12,Featured Research Session,The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Effect of Ischemic Preconditioning and Mitochondrial K_ Channel Openers on Chronic Left Ventricular Remodeling in the Ischemic-Reperfused Rat Heart
PJ-430 Domain2114-2149 within Ryanodine Receptor (RyR) Plays a Key Role for Stabilization of RyR2 in Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT)(PJ073,Arrhythmia, Others (Basic) 1 (A),Poster Session (Japanese),The 73rd Annual Scientific M
PJ-073 Protein Phosphatase 1 Beta is Most Significant Subunit in the Longitudinal Sarcoplasmic Reticulum and Controles Phospholamban Phosphorylation in Cardiomyocytes(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd
PJ-071 In Vivo Knockdown of Protein Phosphatase 1β Augments Cardiac Contracitility in Mice(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
PJ-068 Abnormally Tight Domain-Domain Interaction at Mutation Site could be a Primary Cause of Catecholaminergic Polymorphic Ventricular Tachycardia(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd Annual Scientific
5 Decreased affinity of calmodulin binding to RyR2 may cause leaky channel in CPVT-associated mutation(Young Investigator's Award for International Students Finalists Lecture (YIA International),The 73rd Annual Scientific Meeting of The Japanese Circulati
3 Correction of RyR2 Defects as a New Therapeutic Strategy against Heart Failure(Novel Therapeutic Strategies against Heart Failure,KSC-JCS Joint Symposium (KSC-JCS Joint Symposium) (I),The 73rd Annual Scientific Meeting of The Japanese Circulation Societ
Defective Ca^ Cycling as a Key Pathogenic Mechanism of Heart Failure(Molecular Mechanisms)
FRS-018 Marked Increase in Spontaneous Ca2+ Sparks In Response To β-stimulation As A Critical Cause Of Catecholaminergic Polymorphic Ventricular Tachycardia(New Insights into Basic Mechanism of Arrhythmia(A),Featured Research Session,The 72nd Annual Scien
FRS-017 Enhanced Sensitivity of the Cardiac Ryanodine Receptor to Activation by Luminal Ca^ as a Primary Cause of CPVT(New Insights into Basic Mechanism of Arrhythmia(A),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circ
4 Phospho-regulation of Sarcoplasmic Reticulum Microdomain in Heart Failure(Symposium 7 (SY-07) (M) Molecular Mechanism and Novel Therapeutic Target in Heart Failure,Special Program,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PJ-765 Scavenging Free Radicals by Low-dose Carvedilol Prevents Redox-dependent Ca^ Leak via Stabilization of Ryanodine Receptor in Heart Failure(Development and differentiation-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society
OJ-249 Interruption of Specific Domain-domain Interaction within Ryanodine Receptor Mediates Stabilization of Ryanodine Receptor in Failing Hearts(Heart failure, basic-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
AT_1 Receptor Antagonist Restores Cardiac Ryanodine Receptor Function, Rendering Isoproterenol-Induced Failing Heart Less Susceptible to Ca^-Leak Induced by Oxidative Stress(Experimental Investigation)
OJ-295 Effect of Tautomycin, a Chemical Inhibitor of Protein Phosphatase 1, on Cellular Contractility and Calcium Transient in Adult Rat Cardiomyocytes(Cardiac function, basic/clinical-2 (M) OJ50,Oral Presentation (Japanese),The 70th Anniversary Annual Sc
Tongue Muscle-Derived Stem Cells Express Connexin 43 and Improve Cardiac Remodeling and Survival After Myocardial Infarction in Mice
PE-164 Defective Inter-domain Interaction may Cause Spontaneous Ca^ Release via Reduced Affinity of Calmodulin Binding to RyR2 in Failing Hearts(PE028,Heart Failure, Basic (M),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese
Defective Regulation of Inter-domain Interactions within the Ryanodine Receptor Plays a Key Role in the Pathogenesis of Heart Failure (Excitation-contraction Coupling/Ion Channel 2 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Societ
Effects of In Vivo Gene Transfer of Fibroblast Growth Factor-2 on Cardiac Function and Collateral Vessel Formation in the Microembolized Rabbit Heart
OJ-249 Tenascin-C is Tissue Protective during the Development of Abdominal Aortic Aneurysm in Mice(OJ42,Peripheral Circulation/Vascular Disease (Pathophysiology, Basic) (H),Oral Presentation (Japanese),The 73rd Annual Scientific Meeting of The Japanese Ci
FRS-075 Chronic Inhibition of Apoptosis Signal-regulating Kinase 1 (ASK1) by Myocardial Gene Transfer Suppressed Progression of Heart Failure in Genetic Cardiomyopathy(Heart Failure Research (M) FRS15,Featured Research Session,The 70th Anniversary Annual
OE-310 Specific Inhibition of Sarcoplasmic Reticulum-associated Protein Phosphatase 1 by Inhibitor-2 Confers a Novel Approach for Treatment of Heart Failure(Heart failure, basic-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Candesartan Prevents PKA-hyperphosphorylation, being Less Susceptible to Ca2+ Leak through Ryanodine Receptor in Isoproterenol-induced Failing Rats(Heart Failure, Basic 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OJ-016 Defective Regulation of Inter-Domain Interaction within Ryanodine Receptor as a Cause of Heart Failure(Excitation-Contraction Coupling/Ion Channel (A) : OJ3)(Oral Presentation (Japanese))
PJ-707 Regulation of Cardiac Regeneration by ACE Inhibitor Following Donor Heart Myocardial Infarction with Heterotopic Transplant-Coronary Ligation Model(Regeneration (angionenesis/myocardial regeneration)-7 (M) PJ119,Poster Session (Japanese),The 70th A
PE-190 Involvement of JNK2 in Mechanosensing and Proinflammatory Signaling during Development of Aortic Aneurysm(PE032,Peripheral Circulation/Vascular Disease (Pathophysiology, Basic) (H),Poster Session (English),The 73rd Annual Scientific Meeting of The
PE-179 JNK Activation Causes Endoplasmic Reticulum Stress Response in Adipocytes to Suppress The Adiponectin Secretion In Vivo(Diabetes/Obesity/Metabolic syndrome(05)(H),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulatio
PE-115 Critical Role of JNK2 for Development of Abdominal Aortic Aneurysm in Mice(Peripheral circulation/Vascular disease(03)(H),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OJ-092 Protein Phosphatase 1 β is the Major Phospholamban Phosphatase that Negatively Regulates Cardiac Contraction and Relaxation.(Heart failure, basic(02)(M),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Soci
2 Critical Role of JNK in Chronic Inflammation of Abdominal Aortic Aneurysms(Chronic Inflammation and Cardiovascular Diseases,Roundtable Discussion 7 (RT-07) (H),Special Program,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
Myocardial Free Fatty Acid (FFA) Metabolism in Several Heart Diseases
The Mechanism Of Myocardial Ischemia that happened at the Recovery Phase of Exercise Tolerance Test.
OE-175 Stabilization of Extracellular Matrix Counteracts Proinflammatory Signaling and Prevents Progression of Abdominal Aortic Aneurysm(Molecular biology, vascular-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
FRS-034 Statins Reduce NF-κ B Activation and Chemokine Secretion in Human Abdominal Aortic Aneurysm Wall(Pathophysiology of Cardiovascular Disease, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-134 Inhibition of c-Jun N-terminal Kinase Causes Regression of Abdominal Aortic Aneurysm in Murine Models(Peripheral circulation-1 (H) OE23,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
OE-096 c-Jun N-terminal Kinase Is Required for Development of Abdominal Aortic Aneurysm In Vivo(Molecular Biology, Vascular 1 (H) : OE12)(Oral Presentation (English))
Significance of alpha 2 Adrenergic Activity on myocardial Function when the coronary Flow Reserve Is Abolished.
-0934- EFFECTS OF ATP SENSITIVE POTASSIUM CHANNEL(K-ATP)ON ISCHEMIA INDUCED INTERSTITIAL NOREPINEPHRINE(NE)RELEASE(PROCEEDINGS OF THE 59th ANNUAL SCIENTIFIC MEETING OF THE JAPANESE CIRCULATION SOCIETY)
Cardiac Gene Transfer of Protein Phosphatase 1 Inhibitor-2 Restores Left Ventricular Function in Progressive Hamster Cardiomyopathy(Heart Failure, Basic 2 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PJ-228 Protein Phosphatase Dysregulation and Its Effect on Phosphorylation of Ca^ Regulatory Proteins in Hamster Cardiomyopathy(Heart Failure, Basic 2 (M) : PJ38)(Poster Session (Japanese))
FRS-019 Role of Cytosolic Regulatory Subunits of Protein Phosphatase 1 on Cell Shortening and Ca2+ Homeostasis in Isolated Adult Rat Cardiomyocytes(Cardiac Hypertrophy (M) : FRS3)(Featured Research Session (English))
OE-315 Protein Phosphatase 1β is the Major Phospholamban Phosphatase in Adult Rat Cardiomyocytes(Heart failure, basic-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
4 The Essential Role of c-Jun N-Terminal Kinase in Destructive Vascular Remodeling(New Insight and Therapeutic Strategy for Plaque Stabilization, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-116 Id2 Regulates Post-Developmental Phenotype of Vascular Smooth Muscle Cells in Atherosclerosis In Vivo(Atherosclerosis, Basic 1 (IHD) : OE14)(Oral Presentation (English))
Tenascin-C is expressed in abdominal aortic aneurysm tissue with an active degradation process

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