Nephrotic Syndrome and End-stage Kidney Disease Accompanied by Bicytopenia due to Copper Deficiency
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概要
- 論文の詳細を見る
A 69-year-old man presented with proteinuria and hematuria. He had received total parenteral nutrition for massive small bowel resection. However, due to the iatrogenic lack of trace elements for the next four years, he developed severe copper-deficiency anemia and neutropenia. In addition, his proteinuria and kidney dysfunction worsened concurrently with the development of nephrotic syndrome and end-stage kidney disease. After receiving trace elements, the patient's anemia and neutropenia improved, and the anuria dramatically resolved. Copper-containing enzymes, including ceruloplasmin have an antioxidant activity. In patients with various types of glomerular injuries, the ceruloplasmin expression is known to be increased. Copper deficiency can worsen nephrotic syndrome by decreasing the ceruloplasmin activity, which protects the glomeruli.
- 一般社団法人 日本内科学会の論文
著者
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Takata Takuma
Departmanets Of Structural Pathology Institute Of Nephrology Graduate School Of Medical And Dental S
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Ito Yumi
Division Of Clinical Nephrology And Rheumatology Niigata University Graduate School Of Medical And D
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Narita Ichiei
Division Of Clinical Nephrology And Intensive Care Medicine Niigata University Graduate School Of Me
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Yamazaki Hajime
Department Of Anesthesiology Dokkyo University School Of Medicine
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Watanabe Yasuo
Department Of Agricultural Chemistry College Of Agriculture Ehime University
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Imai Naofumi
Division Of Clinical Nephrology And Rheumatology Niigata University Graduate School Of Medical And Dental Sciences
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Ito Yumi
Division of Clinical Nephrology and Rheumatology, Niigata University Graduate School of Medical and Dental Sciences, Japan
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Sakamaki Yuichi
Division of Clinical Nephrology and Rheumatology, Niigata University Graduate School of Medical and Dental Sciences, Japan
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Goto Kei
Division of Clinical Nephrology and Rheumatology, Niigata University Graduate School of Medical and Dental Sciences, Japan
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