AN EXPERIMENTAL STUDY ON THE CAUSE OF ABNORMAL HYPERMOTILITY OF THE PYLORIC PORTION AFTER GASTRIC TRANSECTION

概要

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It is generally known that the transection anastomosis of the stomach results in the hypermotility of the pyloric portion. The cause of this hypermotility, whether it could be rendered to the transection of the gastric wall or to the simultaneous vagal. denervation distal to the transection line has been a matter of dispute. The present experiment was designed to study the motility changes of the canine stomach with the use of electromygoraphy after transection-anastomosis of the stomach with or without preservation of the vagus nerves.<BR>1) Transection-anastomosis of the stomach with vagal preservation elicited abnormal hypermotility of the pyloric portion; e.g., prolonged discharge interval of normo-peristalses, faster propagation velocity and frequent occurrence of anti-peristaltic discharges.<BR>2) Transection-anastomosis of the stomach without vagal preservation yielded almost the same results as mentioned above.<BR>3) Bilateral truncal vagotomy alone failed to show any noticeable changes in gastric motility. Consecutive transection-anastomosis on the vagotomized stomach resulted in the same changes of the pyloric; portion as seen after transection- anastomosis with vagal preservation.<BR>These results suggest that abnormal hypermotility of the pyloric; portion seen after transection-anastomosis can be attributed to the effect of transection of the gastric wall per se but not to the one of vagotomy.
日本平滑筋学会の論文