Origin of Diabetes Insipidus and Hypernatremia
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概要
- 論文の詳細を見る
There are few studies dealing with the pathophysiology in the central mechanism of water and electrolytes control. The osmoreceptors have been concerned in the region of the supraoptic and paraventricular nuclei (SON, PVN) which are also the area where ADH is formed. Investigations were made on the role of osmoreceptors in ADH-releasing disorder and in experimentally induced diabetes insipidus in the cat and on the involvement of the lateral hypothalamus in these disorders as well as in hypernatremia resulting from disorders of osmoreceptors and the drinking or thirst centers. Daily intake and output, pre- and postoperative electrolytes and osmolarity in the serum and urine, plasma ADH and aldosterone were measured in three groups (normal, diabetes insipidus and hypernatremia cats). Experimental diabetes insipidus was induced by the SON lesion on one side, while mild diabetes insipidus with concurrent development of hypernatremia was produced by the SON lesion and an injury to the lateral hypothalamus.
- 社団法人日本脳神経外科学会の論文
著者
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Mori Kazuo
Department Of Electrical And Electronic Engineering Graduate School Of Engineering Mie University
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Iwayama Kaoru
Department Of Neurosurgery Nagasaki University Faculty Of Medicine
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Iwayama Kaoru
Department Of Neurosurgery Nagasaki University School Of Medicine
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TERAMOTO Shigeyoshi
Department of Neurosurgery, National Nagasaki Central Hospital
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YONEKURA Masatoshi
Department of Neurosurgery, Nagasaki University School of Medicine
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Yonekura Masatoshi
Department Of Neurosurgery Nagasaki University School Of Medicine
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Teramoto Shigeyoshi
Department Of Neurosurgery Nagasaki University School Of Medicine:nationa Ohmura Hospital
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Teramoto Shigeyoshi
Department Of Neurosurgery National Nagasaki Central Hospital
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Mori Kazuo
Department Of Clinical Pharmacy Oita Medical University
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