Genetic susceptibility or resistance to autoimmune encephalomyelitis in MHC congenic mice is associated with differential production of pro- and anti-inflammatory
スポンサーリンク
概要
著者
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Weiner H
Harvard Medical School Ma Usa
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Weiner Howard
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Weiner Howard
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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Hattori Masakazu
Joslin Diabetes Center
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Kuchroo V
Harvard Medical School Ma Usa
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Kuchroo Vijay
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Hancock Wayne
Leukosite
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SLAVIN Anthony
Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School
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MARON Ruth
Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School
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Maron R
Harvard Medical School Ma Usa
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Maron Ruth
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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Slavin Anthony
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Slavin Anthony
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
関連論文
- Recovery from experimental allergic encephalomyelitis is TGF-β dependent and associated with increases in CD4^+LAP^+ and CD4^+CD25^+ T cells
- IL-10 is involved in the suppression of experimental autoimmune encephalomyelitis by CD25^+ CD4^+ regulatory T cells
- Mucosal administration of IL-10 enhances oral tolerance in autoimmune encephalomyelitis and diabetes
- Genetic susceptibility or resistance to autoimmune encephalomyelitis in MHC congenic mice is associated with differential production of pro- and anti-inflammatory
- Induction of T^h2 cell differentiation in the primary immune response: dendritic cells isolated from adherent cell culture treated with IL-10 prime naive CD4^+T cells to secrete IL-4
- CTLA-4 is required for the induction of high dose oral tolerance
- Identification of genetic loci associated with paralysis, inflammation and weight loss in mouse experimental autoimmune encephalomyelitis
- Modulation of CD4 co-receptor limits spontaneous autoimmunity when high-affinity transgenic TCR specific for self-antigen is expressed on a genetically resistant background
- Changes in the strength of co-stimulation through the B7/CD28 pathway alter functional T cell responses to altered peptide ligands
- Oral tolerance to copolymer 1 in myelin basic protein (MBP) TCR transgenic mice : cross-reactivity with MBP-specific TCR and differential induction of anti-inflammatory cytokines
- Peripheral tolerance of T_h2 lymphocytes induced by continuous feeding of ovalbumin
- Tuning T cell activation threshold and effector function with cross-reactive peptide ligands
- A chimeric T cell receptor with super-signaling properties
- T_h17 cells : from precursors to players in inflammation and infection