A chimeric T cell receptor with super-signaling properties
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概要
- 論文の詳細を見る
- 2004-07-01
著者
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Kuchroo Vijay
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Backstrom B.
Malaghan Institute Of Medical Research
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Petersen Troels
Malaghan Institute Of Medical Research
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GULLAND Sven
Malaghan Institute of Medical Research
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BETTELLI Estelle
Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School
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PALMER Ed
Laboratory of Transplantation Immunology and Nephrology, Department of Research, University Hospital
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Bettelli Estelle
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Palmer Ed
Laboratory Of Transplantation Immunology And Nephrology Department Of Research University Hospital B
関連論文
- Recovery from experimental allergic encephalomyelitis is TGF-β dependent and associated with increases in CD4^+LAP^+ and CD4^+CD25^+ T cells
- IL-10 is involved in the suppression of experimental autoimmune encephalomyelitis by CD25^+ CD4^+ regulatory T cells
- Genetic susceptibility or resistance to autoimmune encephalomyelitis in MHC congenic mice is associated with differential production of pro- and anti-inflammatory
- Modulation of CD4 co-receptor limits spontaneous autoimmunity when high-affinity transgenic TCR specific for self-antigen is expressed on a genetically resistant background
- Changes in the strength of co-stimulation through the B7/CD28 pathway alter functional T cell responses to altered peptide ligands
- IL-5-deficient mice are susceptible to experimental autoimmune encephalomyelitis
- Tuning T cell activation threshold and effector function with cross-reactive peptide ligands
- Induction of experimental autoimmune encephalomyelitis in the absence of c-Jun N-terminal kinase 2
- A chimeric T cell receptor with super-signaling properties
- T_h17 cells : from precursors to players in inflammation and infection