T_h17 cells : from precursors to players in inflammation and infection

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概要

• 論文の詳細を見る
• 2009-05-01

著者

• Kuchroo Vijay

  Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School

• Awasthi Amit

  Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School

関連論文

• Recovery from experimental allergic encephalomyelitis is TGF-β dependent and associated with increases in CD4^+LAP^+ and CD4^+CD25^+ T cells
• IL-10 is involved in the suppression of experimental autoimmune encephalomyelitis by CD25^+ CD4^+ regulatory T cells
• Genetic susceptibility or resistance to autoimmune encephalomyelitis in MHC congenic mice is associated with differential production of pro- and anti-inflammatory
• Modulation of CD4 co-receptor limits spontaneous autoimmunity when high-affinity transgenic TCR specific for self-antigen is expressed on a genetically resistant background
• Changes in the strength of co-stimulation through the B7/CD28 pathway alter functional T cell responses to altered peptide ligands
• Tuning T cell activation threshold and effector function with cross-reactive peptide ligands
• A chimeric T cell receptor with super-signaling properties
• T_h17 cells : from precursors to players in inflammation and infection

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