Identification of genetic loci associated with paralysis, inflammation and weight loss in mouse experimental autoimmune encephalomyelitis
スポンサーリンク
概要
著者
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Weiner Howard
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Weiner Howard
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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Weiner Howard
Center For Neurologic Diseases Department Of Medicine Brighham & Women's Hospital
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Seidman E
Department Of Genetics Howard Hughes Medical Institute Harvard Medical School
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ENCINAS Jeffrey
Center for Neurologic Diseases, Department of Medicine, Brighham & Women's Hospital
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LEEA Marjorie
Department of Neurology, Harvard Medical School, Boston
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SOBEL Raymond
Department of Pathology, Stanford University School of Medicine and Laboratory Service, Veterans Hea
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SYMONOWICZ Cammie
E. K. Shriver Center
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SEIDMAN J.
Department of Genetics, Howard Hughes Medical Institute, Harvard Medical School
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KUCHROO Vijoy
Center for Neurologic Diseases, Department of Medicine, Brighham & Women's Hospital
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Seidman J.
Department Of Genetics Howard Hughes Medical Institute Harvard Medical School
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Sobel Raymond
Department Of Pathology Stanford University School Of Medicine And Laboratory Service Veterans Healt
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Encinas Jeffrey
Center For Neurologic Diseases Department Of Medicine Brighham & Women's Hospital
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Leea Marjorie
Department Of Neurology Harvard Medical School Boston
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Kuchroo Vijoy
Center For Neurologic Diseases Department Of Medicine Brighham & Women's Hospital
関連論文
- Recovery from experimental allergic encephalomyelitis is TGF-β dependent and associated with increases in CD4^+LAP^+ and CD4^+CD25^+ T cells
- IL-10 is involved in the suppression of experimental autoimmune encephalomyelitis by CD25^+ CD4^+ regulatory T cells
- Mucosal administration of IL-10 enhances oral tolerance in autoimmune encephalomyelitis and diabetes
- Genetic susceptibility or resistance to autoimmune encephalomyelitis in MHC congenic mice is associated with differential production of pro- and anti-inflammatory
- Induction of T^h2 cell differentiation in the primary immune response: dendritic cells isolated from adherent cell culture treated with IL-10 prime naive CD4^+T cells to secrete IL-4
- CTLA-4 is required for the induction of high dose oral tolerance
- Identification of genetic loci associated with paralysis, inflammation and weight loss in mouse experimental autoimmune encephalomyelitis
- Oral tolerance to copolymer 1 in myelin basic protein (MBP) TCR transgenic mice : cross-reactivity with MBP-specific TCR and differential induction of anti-inflammatory cytokines
- Peripheral tolerance of T_h2 lymphocytes induced by continuous feeding of ovalbumin