Oral tolerance to copolymer 1 in myelin basic protein (MBP) TCR transgenic mice : cross-reactivity with MBP-specific TCR and differential induction of anti-inflammatory cytokines
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概要
- 論文の詳細を見る
- 2002-02-01
著者
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Weiner Howard
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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Maron Ruth
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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Slavin Anthony
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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Komagata Y
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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HOFFMANN Ethan
Center for Neurologic Disease, Brigham and Women's Hospital, Harvard Medical School
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KOMAGATA Yoshinori
Center for Neurologic Disease, Brigham and Women's Hospital, Harvard Medical School
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Hoffmann Ethan
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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KOMAGATA Yoshinori
Center for Neurologic Disease, Brigham and Women's Hospital, Harvard Medical School
関連論文
- Recovery from experimental allergic encephalomyelitis is TGF-β dependent and associated with increases in CD4^+LAP^+ and CD4^+CD25^+ T cells
- IL-10 is involved in the suppression of experimental autoimmune encephalomyelitis by CD25^+ CD4^+ regulatory T cells
- Mucosal administration of IL-10 enhances oral tolerance in autoimmune encephalomyelitis and diabetes
- Genetic susceptibility or resistance to autoimmune encephalomyelitis in MHC congenic mice is associated with differential production of pro- and anti-inflammatory
- Induction of T^h2 cell differentiation in the primary immune response: dendritic cells isolated from adherent cell culture treated with IL-10 prime naive CD4^+T cells to secrete IL-4
- CTLA-4 is required for the induction of high dose oral tolerance
- Identification of genetic loci associated with paralysis, inflammation and weight loss in mouse experimental autoimmune encephalomyelitis
- Oral tolerance to copolymer 1 in myelin basic protein (MBP) TCR transgenic mice : cross-reactivity with MBP-specific TCR and differential induction of anti-inflammatory cytokines
- Peripheral tolerance of T_h2 lymphocytes induced by continuous feeding of ovalbumin