Peripheral tolerance of T_h2 lymphocytes induced by continuous feeding of ovalbumin
スポンサーリンク
概要
著者
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Weiner Howard
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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Weiner Howard
Center For Neurological Diseaese Brigham And Women's Hospital Harvard Medical School
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Fishman-lobell Jacqueline
Center For Neurological Diseaese Brigham And Women's Hospital Harvard Medical School
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Melamed Doron
Department Of Animal Sciences Hebrew University Of Jerusalem
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UNI Zehava
Department of Animal Sciences, Hebrew University of Jerusalem
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FRIEDMAN Aharon
Department of Animal Sciences, Hebrew University of Jerusalem
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Uni Zehava
Department Of Animal Sciences Hebrew University Of Jerusalem
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Friedman Aharon
Department Of Animal Sciences Hebrew University Of Jerusalem
関連論文
- Recovery from experimental allergic encephalomyelitis is TGF-β dependent and associated with increases in CD4^+LAP^+ and CD4^+CD25^+ T cells
- IL-10 is involved in the suppression of experimental autoimmune encephalomyelitis by CD25^+ CD4^+ regulatory T cells
- Mucosal administration of IL-10 enhances oral tolerance in autoimmune encephalomyelitis and diabetes
- Genetic susceptibility or resistance to autoimmune encephalomyelitis in MHC congenic mice is associated with differential production of pro- and anti-inflammatory
- Induction of T^h2 cell differentiation in the primary immune response: dendritic cells isolated from adherent cell culture treated with IL-10 prime naive CD4^+T cells to secrete IL-4
- CTLA-4 is required for the induction of high dose oral tolerance
- Identification of genetic loci associated with paralysis, inflammation and weight loss in mouse experimental autoimmune encephalomyelitis
- Increased plasma cell frequency and accumulation of abnormal syndecan-1^ T-cells in Igμ-deficient/lpr mice
- Oral tolerance to copolymer 1 in myelin basic protein (MBP) TCR transgenic mice : cross-reactivity with MBP-specific TCR and differential induction of anti-inflammatory cytokines
- Peripheral tolerance of T_h2 lymphocytes induced by continuous feeding of ovalbumin
- A truncated IFN-regulatory factor-8＼IFN consensus sequence-binding protein acts as dominant-negative, interferes with endogenous protein-protein interactions and leads to apoptosis of immune cells
- TOLL-like receptor ligands stimulate aberrant class switch recombination in early B cell precursors