Increased plasma cell frequency and accumulation of abnormal syndecan-1^<plus> T-cells in Igμ-deficient/lpr mice
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概要
- 論文の詳細を見る
- 2003-09-01
著者
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KARIN Nathan
Department of Immunology, Bruce Rappaport Faculty of Medicine
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Wildbaum Gizi
Department Of Immunology Bruce Rappaport Faculty Of Medicine Technion-israel Institute Of Technology
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SEAGAL Jane
Department of Immunology, Bruce Rappaport Faculty of Medicine, Technion-Israel Institute of Technolo
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LEIDER Nira
Department of Immunology, Bruce Rappaport Faculty of Medicine, Technion-Israel Institute of Technolo
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MELAMED Doron
Department of Immunology, Bruce Rappaport Faculty of Medicine, Technion-Israel Institute of Technolo
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Seagal Jane
Department Of Immunology Bruce Rappaport Faculty Of Medicine Technion-israel Institute Of Technology
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Leider Nira
Department Of Immunology Bruce Rappaport Faculty Of Medicine Technion-israel Institute Of Technology
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Karin Nathan
Department Of Immunology Bruce Rappaport Faculty Of Medicine Technion-israel Institute Of Technology
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Karin Nathan
Department Of Immunology Bruce Rappaport Faculty Of Medicine
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Melamed Doron
Department Of Immunology Bruce Rappaport Faculty Of Medicine Technion-israel Institute Of Technology
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Melamed Doron
Department Of Animal Sciences Hebrew University Of Jerusalem
関連論文
- Expansion of neonatal tolerance to self in adule life: l. The role of a bacterial adjuvant in tolerance spread
- Increased plasma cell frequency and accumulation of abnormal syndecan-1^ T-cells in Igμ-deficient/lpr mice
- Peripheral tolerance of T_h2 lymphocytes induced by continuous feeding of ovalbumin
- Expansion of neonatal tolerance to self in adult life: ll. Tolerance preferentially spreads in an intramolecular manner
- A truncated IFN-regulatory factor-8＼IFN consensus sequence-binding protein acts as dominant-negative, interferes with endogenous protein-protein interactions and leads to apoptosis of immune cells
- Generation and selection of an IgG-driven autoimmune repertoire during B-lymphopoiesis in Igμ-deficient/Ipr mice
- TOLL-like receptor ligands stimulate aberrant class switch recombination in early B cell precursors