Weiner H | Harvard Medical School Ma Usa
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概要
関連著者
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Weiner H
Harvard Medical School Ma Usa
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Weiner Howard
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Weiner Howard
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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Kuchroo Vijay
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Kuchroo V
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Zhang Xianghua
Department Of Immunology National Jewish Medical And Research Center And University Of Colorado Scho
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Reddy Jayagopala
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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ZHANG Xingmin
Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School
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SLAVIN Anthony
Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School
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MARON Ruth
Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School
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Maron R
Harvard Medical School Ma Usa
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Maron Ruth
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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Slavin Anthony
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Slavin Anthony
Center For Neurologic Disease Brigham And Women's Hospital Harvard Medical School
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Zhang Xingmin
Center For Neurologic Diseases Harvard Medical School
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Frenkel Dan
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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SAKAGUCHI Shimon
Department of Experimental Pathology, Institute for Frontier Medical Sciences, Kyoto University
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Hattori Masakazu
Joslin Diabetes Center
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Sakaguchi Shimon
Department Of Experimental Pathology Institute For Frontier Medical Sciences
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Kuchroo V
Harvard Medical School Ma Usa
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Ochi Hirofumi
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Hancock Wayne
Leukosite
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Sakaguchi S
Department Of Immunopathology Tokyo Metropolitan Institute Of Gerontology
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KOLDZIC Djordje
Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School
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IZIKSON Leonid
Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School
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NAZARENO Remedios
Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School
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LIU Liming
Center for Neurologic Diseases, Brigham and Women's Hospital and Harvard Medical School
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RICH Benjamin
Harvard Skin Research Center, Brigham and Women's Hospital and Harvard Medical School
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INOBE Jun-ichi
Center for Neurologic Diseases, Brigham and Women's Hospital and Harvard Medical School
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CHEN Wanjun
Center for Neurologic Diseases, Brigham and Women's Hospital and Harvard Medical School
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Liu L
Hubei Univ. Wuhan Chn
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Izikson Leonid
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Rich Benjamin
Harvard Skin Research Center Brigham And Women's Hospital And Harvard Medical School
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Nazareno Remedios
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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Inobe J
Center For Neurologic Diseases Brigham And Women's Hospital And Harvard Medical School
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Chen W
Center For Neurologic Diseases Brigham And Women's Hospital And Harvard Medical School
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Koldzic Djordje
Center For Neurologic Diseases Brigham And Women's Hospital Harvard Medical School
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CHEN Wanjun
Center for Neurologic Diseases, Brigham and Women's Hospital and Harvard Medical School
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INOBE Jun-ichi
Center for Neurologic Diseases, Brigham and Women's Hospital and Harvard Medical School
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LIU Liming
Center for Neurologic Diseases, Brigham and Women's Hospital and Harvard Medical School
著作論文
- Recovery from experimental allergic encephalomyelitis is TGF-β dependent and associated with increases in CD4^+LAP^+ and CD4^+CD25^+ T cells
- IL-10 is involved in the suppression of experimental autoimmune encephalomyelitis by CD25^+ CD4^+ regulatory T cells
- Mucosal administration of IL-10 enhances oral tolerance in autoimmune encephalomyelitis and diabetes
- Genetic susceptibility or resistance to autoimmune encephalomyelitis in MHC congenic mice is associated with differential production of pro- and anti-inflammatory
- Induction of T^h2 cell differentiation in the primary immune response: dendritic cells isolated from adherent cell culture treated with IL-10 prime naive CD4^+T cells to secrete IL-4