Yanagihara Nobuyuki | Department of Pharmacology, School of Medicine, University of Occupational and Environmental Health

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著作論文

PJ-731 Genetic Disruption of All Nitric Oxide Synthase Isoforms Causes Accelerated Renal Lesion Formation in Mice in Vivo(PJ123,Kidney/Renal Circulation/CKD 6 (H),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Soc
10P-E-1 Algorithm for Blood Vessel Cleaning by United Gene Expression Programming(Room E International session)
Nitric Oxide Synthases and Cardiovascular Diseases : Insights From Genetically Modified Mice
PE-259 Mechanisms for the Different Functions of Endothelial Nitric Oxide Synthases System between Conduit and Resistance Arteries in Mice(PE044,Molecular Biology (M),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation
PE-188 Characterization of Vascular Function in Mice Lacking Entire Nitric Oxide Synthase System(PE032,Peripheral Circulation/Vascular Disease (Pathophysiology, Basic) (H),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circula
PJ-095 Endothelium-Dependent Hyperpolarizations are Totally Dependent on Endothelial Nitric Oxide Synthases System : Lessons from Mice Lacking All Nitric Oxide Synthase Isoforms(Endothelium(03)(IHD),Poster Session(Japanese),The 72nd Annual Scientific Meet
Interleukin (IL)-4 and IL-13 Inhibit the Differentiation of Murine Osteoblastic MC3T3-E1 Cells
Stimulatory Effect of Interleukin-1α on Proliferation through a Ca^/Calmodulin dependent Pathway of a Human Thyroid Carcinoma Cell Line, NIM 1
Nephrogenic Diabetes Insipidus in Mice Lacking All Nitric Oxide Synthase Isoforms(Endothelial Function (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Vasculoprotective Role of Neuronal Nitric Oxide Synthase in Vascular Lesion Formation in Rats
Direct Evidence for the NO-Independent Actions of Asymmetric Dimethylarginine in Vascular Lesion Formation in Vivo
OE-359 Severe Dyslipidemia and Lipid-Rich Atherosclerotic Lesion Formation in Mice Lacking All Nitric Oxide Synthases Fed a High-Cholesterol Diet(OE61,Lipid Disorders (H),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circu
OE-197 Left Ventricular Hypertrophy and Diastolic Dysfunction in Mice Lacking All Nitric Oxide Synthase Isoforms(OE34,Cardiac Function (Basic) (M),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
OE-012 Genetic Disruption of Entire Nitric Oxide Synthase System Causes Activation of Renin-Angiotensin-Aldosterone Axis in Mice in Vivo(OE02,ACS/AMI (Basic) (IHD),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation
OJ-088 Abnormal Low-Density Lipoprotein Metabolism in Mice Lacking All Nitric Oxide Synthase Isoforms(Lipid disorders(02)(H),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OE-271 Important Role of the Renin-Angiotensin System in the Pathogenesis of Myocardial Infarction in Mice Lacking All Nitric Oxide Synthase Isoforms(Acute myocardial infarction, basic(01)(IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting
OE-093 Statin Treatment Ameliorates Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthase Isoforms(Diabetes/Obesity/Metabolic syndrome(01)(H),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
FRS-036 Gender Difference of Arteriosclerotic Cardiovascular Diseases in Mice Lacking All Nitric Oxide Synthase Isoforms(Peripheral Vascular Disease(H),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PE-207 Left Ventricular Hypertrophy and Diastolic Dysfunction in Mice Lacking All Nitric Oxide Synthases(Cardiac hypertrophy, basic/clinical-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PE-042 Dyslipidemia, Lipid-Rich Atherosclerotic Lesion Formation, and Sudden Death in Mice Lacking All Nitric Oxide Synthases Fed a High-Cholesterol Diet(Atherosclerosis, basic-5, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OJ-214 Abnormal Glucose Metabolism in Mice Lacking All Nitric Oxide Synthase Isoforms(Genetics/Genetically engineered models/Gene therapy-2(H/M), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-356 Endothelium-Dependent Hyperpolarizations are Totally Dependent on Endothelial Nitric Oxide Synthases System : Lessons from Mice Lacking All Nitric Oxide Synthase Isoforms(Endothelium-1, The 71st Annual Scientific Meeting of the Japanese Circulation
OE-281 Hyper-Low-Density-Lipoproteinemia in Mice Lacking All Nitric Oxide Synthases(Lipid disorders-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Long-Term Treatment With Imidapril but Not With Nifedipine Enhances Plasma NOx Concentration in Patients With Essential Hypertension
Treatment with imidapril but not with nifedipine increases plasma NOx concentrations in patients with essential hypertension
Long-Term Blockade of Angiotensin-Converting Enzyme Ameliorates Dobutamine-Induced Myocardial Ischemia in Patients with Coronary Artery Disease
Long-Term Treatment With Perindopril Ameliorates Dobutamine-Induced Myocardial Ischemia in Patients With Coronary Artery Disease
Direct Inhibition of N-Methyl-D-Aspartate (NMDA)-Receptor Function by Antiglaucomatous β-Antagonists
PE-433 Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthase Isoforms(Diabetes/Obesity/Metabolic syndrome-4 (H) PE73,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
OE-199 Genetic Disruption of All Nitric Oxide Synthase Isoforms Markedly Reduces Survival in Mice(Endothelium-1 (H) OE34,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
OE-133 Vasculoprotective Role of the Nitric Oxide Synthases System against Vascular Lesion Formation in Mice in Vivo(Peripheral circulation-1 (H) OE23,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation S
OE-063 Spontaneous Development of Systemic Arteriosclerosis in Mice Lacking All Nitric Oxide Synthase Isoforms(Atherosclerosis, basic-1 (H) OE11,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society
Inhibition by Selenium Compounds of Catecholamine Secretion Due to Inhibition of Ca^ Influx in Cultured Bovine Adrenal Chromaffin Cells
Effect of cAMP-Dependent Protein Kinase on Catecholamine Secretion from Bovine Adrenal Chromaffin Cells
FRS-084 Spontaneous Myocardial Infarction in Mice Lacking All Nitric Oxide Synthase Isoforms(Molecular Biology in Cardiovascular Diseases (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
FRS-021 Long-Term Treatment with an Angiotensin II Type 1 Receptor Blocker Ameliorates Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthases(Diabetes/Obesity (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Development of Genetically Engineered Mice Lacking All Three Nitric Oxide Synthases
cDNA Cloning of Inducer Reactive Protein in Adult T-Cell Leukemia Cells by Applying the Fuzzy Set Cloning Theory for Sequencing Defuzzification
Atorvastatin Upregulates Vascular Neuronal Nitric Oxide Synthase Expression Through the Akt/NF-κB Pathway(Endothelium/NO 4 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Vascular Neuronal Nitric Oxide Synthase Expression is Upregulated in Response to Platelet-Derived Growth Factor Through the MEK/ERK Pathway(Endothelium/NO 4 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Neuronal Nitric Oxide Synthase Is Subject to Expressional Regulation by Inflammatory Stimuli in Vascular Smooth Muscle Cells in Rats
Complete Disruption of All Nitric Oxide Synthase Genes Causes Markedly Accelerated Renal Lesion Formation Following Unilateral Ureteral Obstruction in Mice In Vivo
OJ-163 Genetic Disruption of All Nitric Oxide Synthase Isoforms Abolishes EDHF-Mediated Responses in Mice(Endothelium-2 (H) OJ28,Oral Presentation (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Spontaneous Development of Left Ventricular Hypertrophy and Diastolic Dysfunction in Mice Lacking All Nitric Oxide Synthases
Inhibition by Pregnenolone Sulphate, a Metabolite of the Neurosteroid Pregnenolone, of Voltage-Gated Sodium Channels Expressed in Xenopus Oocytes
Long-Term Treatment With San'o-Shashin-To, a Kampo Medicine, Markedly Ameliorates Cardiac Ischemia-Reperfusion Injury in Ovariectomized Rats via the Redox-Dependent Mechanism

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