Nakashima Yasuhide | Second Department Of Internal Medicine University Of Occupational And Environmental Health School Of

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著作論文

PJ-731 Genetic Disruption of All Nitric Oxide Synthase Isoforms Causes Accelerated Renal Lesion Formation in Mice in Vivo(PJ123,Kidney/Renal Circulation/CKD 6 (H),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Soc
10P-E-1 Algorithm for Blood Vessel Cleaning by United Gene Expression Programming(Room E International session)
Nitric Oxide Synthases and Cardiovascular Diseases : Insights From Genetically Modified Mice
PE-259 Mechanisms for the Different Functions of Endothelial Nitric Oxide Synthases System between Conduit and Resistance Arteries in Mice(PE044,Molecular Biology (M),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation
PE-188 Characterization of Vascular Function in Mice Lacking Entire Nitric Oxide Synthase System(PE032,Peripheral Circulation/Vascular Disease (Pathophysiology, Basic) (H),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circula
PJ-095 Endothelium-Dependent Hyperpolarizations are Totally Dependent on Endothelial Nitric Oxide Synthases System : Lessons from Mice Lacking All Nitric Oxide Synthase Isoforms(Endothelium(03)(IHD),Poster Session(Japanese),The 72nd Annual Scientific Meet
OE-359 Severe Dyslipidemia and Lipid-Rich Atherosclerotic Lesion Formation in Mice Lacking All Nitric Oxide Synthases Fed a High-Cholesterol Diet(OE61,Lipid Disorders (H),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circu
OE-197 Left Ventricular Hypertrophy and Diastolic Dysfunction in Mice Lacking All Nitric Oxide Synthase Isoforms(OE34,Cardiac Function (Basic) (M),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
OE-012 Genetic Disruption of Entire Nitric Oxide Synthase System Causes Activation of Renin-Angiotensin-Aldosterone Axis in Mice in Vivo(OE02,ACS/AMI (Basic) (IHD),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation
OJ-088 Abnormal Low-Density Lipoprotein Metabolism in Mice Lacking All Nitric Oxide Synthase Isoforms(Lipid disorders(02)(H),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OE-271 Important Role of the Renin-Angiotensin System in the Pathogenesis of Myocardial Infarction in Mice Lacking All Nitric Oxide Synthase Isoforms(Acute myocardial infarction, basic(01)(IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting
OE-093 Statin Treatment Ameliorates Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthase Isoforms(Diabetes/Obesity/Metabolic syndrome(01)(H),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
FRS-036 Gender Difference of Arteriosclerotic Cardiovascular Diseases in Mice Lacking All Nitric Oxide Synthase Isoforms(Peripheral Vascular Disease(H),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PE-207 Left Ventricular Hypertrophy and Diastolic Dysfunction in Mice Lacking All Nitric Oxide Synthases(Cardiac hypertrophy, basic/clinical-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PE-042 Dyslipidemia, Lipid-Rich Atherosclerotic Lesion Formation, and Sudden Death in Mice Lacking All Nitric Oxide Synthases Fed a High-Cholesterol Diet(Atherosclerosis, basic-5, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OJ-214 Abnormal Glucose Metabolism in Mice Lacking All Nitric Oxide Synthase Isoforms(Genetics/Genetically engineered models/Gene therapy-2(H/M), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-356 Endothelium-Dependent Hyperpolarizations are Totally Dependent on Endothelial Nitric Oxide Synthases System : Lessons from Mice Lacking All Nitric Oxide Synthase Isoforms(Endothelium-1, The 71st Annual Scientific Meeting of the Japanese Circulation
Direct Inhibition of N-Methyl-D-Aspartate (NMDA)-Receptor Function by Antiglaucomatous β-Antagonists
Spontaneous Development of Left Ventricular Hypertrophy and Diastolic Dysfunction in Mice Lacking All Nitric Oxide Synthases
Complete Disruption of All Nitric Oxide Synthase Genes Causes Markedly Accelerated Renal Lesion Formation Following Unilateral Ureteral Obstruction in Mice In Vivo

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