Morishita Tsuyoshi | Second Department of Internal Medicine, University of Occupational and Environmental Health

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著作論文

Cuff-induced Neointimal Formation is Reduced by Extracellular Superoxide Dismutase(Atherosclerosis, Basic 6 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Hyperhomocysteinemia is Associated With Human Coronary Atherosclerosis Through the Reduction of the Ratio of Endothelium-Bound to Basal Extracellular Superoxide Dismutase
PE-188 Characterization of Vascular Function in Mice Lacking Entire Nitric Oxide Synthase System(PE032,Peripheral Circulation/Vascular Disease (Pathophysiology, Basic) (H),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circula
Nephrogenic Diabetes Insipidus in Mice Lacking All Nitric Oxide Synthase Isoforms(Endothelial Function (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Vasculoprotective Role of Neuronal Nitric Oxide Synthase in Vascular Lesion Formation in Rats
Direct Evidence for the NO-Independent Actions of Asymmetric Dimethylarginine in Vascular Lesion Formation in Vivo
OE-359 Severe Dyslipidemia and Lipid-Rich Atherosclerotic Lesion Formation in Mice Lacking All Nitric Oxide Synthases Fed a High-Cholesterol Diet(OE61,Lipid Disorders (H),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circu
OE-197 Left Ventricular Hypertrophy and Diastolic Dysfunction in Mice Lacking All Nitric Oxide Synthase Isoforms(OE34,Cardiac Function (Basic) (M),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
OJ-088 Abnormal Low-Density Lipoprotein Metabolism in Mice Lacking All Nitric Oxide Synthase Isoforms(Lipid disorders(02)(H),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OE-271 Important Role of the Renin-Angiotensin System in the Pathogenesis of Myocardial Infarction in Mice Lacking All Nitric Oxide Synthase Isoforms(Acute myocardial infarction, basic(01)(IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting
OE-093 Statin Treatment Ameliorates Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthase Isoforms(Diabetes/Obesity/Metabolic syndrome(01)(H),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
FRS-036 Gender Difference of Arteriosclerotic Cardiovascular Diseases in Mice Lacking All Nitric Oxide Synthase Isoforms(Peripheral Vascular Disease(H),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PE-207 Left Ventricular Hypertrophy and Diastolic Dysfunction in Mice Lacking All Nitric Oxide Synthases(Cardiac hypertrophy, basic/clinical-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PE-042 Dyslipidemia, Lipid-Rich Atherosclerotic Lesion Formation, and Sudden Death in Mice Lacking All Nitric Oxide Synthases Fed a High-Cholesterol Diet(Atherosclerosis, basic-5, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OJ-214 Abnormal Glucose Metabolism in Mice Lacking All Nitric Oxide Synthase Isoforms(Genetics/Genetically engineered models/Gene therapy-2(H/M), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-281 Hyper-Low-Density-Lipoproteinemia in Mice Lacking All Nitric Oxide Synthases(Lipid disorders-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Long-Term Treatment With Imidapril but Not With Nifedipine Enhances Plasma NOx Concentration in Patients With Essential Hypertension
Treatment with imidapril but not with nifedipine increases plasma NOx concentrations in patients with essential hypertension
Angiotensin Receptor Blocker (ARB) Restores Coronary Flow Velocity Reserve in Patients with Essential Hypertension-Comparison with Calcium Antagonist(Hypertension, Clinical 7 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Long-Term Blockade of Angiotensin-Converting Enzyme Ameliorates Dobutamine-Induced Myocardial Ischemia in Patients with Coronary Artery Disease
Long-Term Treatment With Perindopril Ameliorates Dobutamine-Induced Myocardial Ischemia in Patients With Coronary Artery Disease
PE-311 Usefulness of Contrast Echocardiography for Evaluation of the Patency of Right Coronary Artery Bypass Grafts(Cardiovascular Surgery/CABG 3 (IHD) : PE53)(Poster Session (English))
PE-433 Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthase Isoforms(Diabetes/Obesity/Metabolic syndrome-4 (H) PE73,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
OE-199 Genetic Disruption of All Nitric Oxide Synthase Isoforms Markedly Reduces Survival in Mice(Endothelium-1 (H) OE34,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
OE-133 Vasculoprotective Role of the Nitric Oxide Synthases System against Vascular Lesion Formation in Mice in Vivo(Peripheral circulation-1 (H) OE23,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation S
OE-063 Spontaneous Development of Systemic Arteriosclerosis in Mice Lacking All Nitric Oxide Synthase Isoforms(Atherosclerosis, basic-1 (H) OE11,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society
FRS-084 Spontaneous Myocardial Infarction in Mice Lacking All Nitric Oxide Synthase Isoforms(Molecular Biology in Cardiovascular Diseases (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
FRS-021 Long-Term Treatment with an Angiotensin II Type 1 Receptor Blocker Ameliorates Metabolic Syndrome in Mice Lacking All Nitric Oxide Synthases(Diabetes/Obesity (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Development of Genetically Engineered Mice Lacking All Three Nitric Oxide Synthases
Atorvastatin Upregulates Vascular Neuronal Nitric Oxide Synthase Expression Through the Akt/NF-κB Pathway(Endothelium/NO 4 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Vascular Neuronal Nitric Oxide Synthase Expression is Upregulated in Response to Platelet-Derived Growth Factor Through the MEK/ERK Pathway(Endothelium/NO 4 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Neuronal Nitric Oxide Synthase Is Subject to Expressional Regulation by Inflammatory Stimuli in Vascular Smooth Muscle Cells in Rats
Spontaneous Development of Left Ventricular Hypertrophy and Diastolic Dysfunction in Mice Lacking All Nitric Oxide Synthases

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