Igarashi Norio | 富山大学医学薬学研究部第2内科

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著作論文

Fluvastatin Attenuates Diabetes-Induced Cardiac Sympathetic Neuropathy in Association With a Decrease in Oxidative Stress
PJ-037 Effects of a Central Sympathoinhibitory Drug, Guanfacine, on Cardiac Metaiodobenzylguanidine Scintigraphy in Patients with Heart Failure(Nuclear Cardiology 3 (I) : PJ7)(Poster Session (Japanese))
Supersensitive Response to Isoproterenol in Patients With Marked Global Reduction of Cardiac Metaiodobenzylguanidine Uptake
Cardiac Sympathetic Nerve Activity Alters Myocardial BMIPP Uptake and Its Clearance
Sympathetic Neuronal Function Is Coupled with the Downregulation of β-Adrenoceptors in Ischemia-Reperfusion Rat Hearts
Cardiac washout rate of MIBG is better able to represent sympathetic nerve activity when compared at a comparable MIBG uptake
PJ-543 Focal Myocardial Perfusion Defects Suggest More Advanced Heart Failure in Patients with Dilated Cardiomyopathy(Nuclear cardiology(09)(I),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
FRS-014 Fluvastatin Restores Impaired Cardiac Sympathetic Neuronal Function through Reduction in Oxidative Stress in Diabetic Rats(Topics in Nuclear Cardiology(I),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circulation Soc
Early Administration of Fluvastatin, but not at the Onset of Ischemia or Reperfusion, Attenuates Myocardial Ischemia-Reperfusion Injury Through the Nitric Oxide Pathway Rather Than Its Antioxidant Property
Influence of β-Adrenoceptor Blockade on the Myocardial Accumulation of Fatty Acid Tracer and Its Intracellular Metabolism in the Heart After Ischemia-Reperfusion Injury
Brief Episode of Myocardial Ischemia Before Prolonged Ischemia Attenuates Cardiac Sympathetic Nerve Injury
PJ-780 Hyperglycemia at Reperfusion Dose Not Exacerbate Myocardial Ischemia-reperfusion Injury(Myocardial ischemia-reperfusion, basic/clinical-6 (IHD) PJ136,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulati
PJ-728 A Combined Therapy of AT_1-receptor Antagonist may Enhance the Plaque-stabilizing Effect of Statin(Atherosclerosis, clinical-15 (H) PJ123,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
PJ-726 Increase in C-Reactive Protein within Coronary Circulation after Stent Implantation Correlate with Plaque Progression at Non-Culprit Lesion : Effect of Fluvastatin(Atherosclerosis, clinical-15 (H) PJ123,Poster Session (Japanese),The 70th Anniversar
FRS-036 Beneficial Effects of Ischemic Preconditioning and Diabetes on Myocardial Fatty Acid Metabolism after Ischemia-reperfusion(Advances in Nuclear Cardiology-2 (I) FRS8,Featured Research Session,The 70th Anniversary Annual Scientific Meeting of the Ja
Ischemia-Induced Norepinephrine Release, but not Norepinephrine-Derived Free Radicals, Contributes to Myocardial Ischemia : Reperfusion Injury
Reduced Cardiac Norepinephrine Release during Ischemia is not Necessarily Accompanied by Attenuation of Sympathetic Neuronal Dysfunction in the Remote Period (Nuclear Cardiology 11(I), The 69th Annual Scientific Meeting of the Japanese Circulation Society
The Genotype of CYP2D6 Determines Cardiac Responsiveness to β-adrenergic Stimulation in patients Treated with Metoprolol, but not with Bisoprolol(Cardiovascular Pharmacology, Basic/Clinical 2 (H), The 69th Annual Scientific Meeting of the Japanese Circula
Improvement of Insulin Signaling Induced by Enalapril Contributes to the Inhibition of Developing Hypertensive Heart Failure of Dahl Rats(Nuclear Cardiology 6 (I), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PJ-550 Early Administration of Fluvastatin, Not at the Onset of Ischemia Or Reperfusion, Reduces Myocardial Infarction Size in Ischemia-Reperfusion Injury(Myocardial Ischemia-Reperfusion, Basic and Clinical 4 (M) : PJ92)(Poster Session (Japanese))
PJ-329 Exaggerated and sustained adrenergic responses to transient stress in heart and kidney of unanesthetized hypertensive rats(Hypertension, basic(03)(H),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PJ-072 A combination of assessment of carotid artery plaque with matrix metalloproteinase-9 may predict coronary vulunerable plaque(CT/DSA(07)(I),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
OJ-063 CYP2D6 Genotype Has little Influences on Plasma Concentrations of Carvediol : A Comparison with Metoprolol and Bisoprolol.(Cardiovascular pharmacology, basic/clinical(02)(H),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japa
OJ-321 Inhibition of Increasing Synaptic Norepinephrine during Ischemia by Valsartan Protects Cardiac Sympathetic Neuronal Function and β-Receptors from Ischemia-Reperfusion Injury(Nuclear Cardiology 7 (I) : OJ38)(Oral Presentation (Japanese))
OJ-318 Beneficial effects of β-adrenergic blocker in ischemia-reperfusion injury might partially result from the inhibition of non-metabolite accumulation of fatty acid(Nuclear Cardiology 7 (I) : OJ38)(Oral Presentation (Japanese))
Effects of Valsartan on the Ischemia-Reperfusion Injury of Cardiac Sympathetic Nerve Function and β-Adrenoceptors
Effect of CYP2D6^*10 on the Pharmacokinetics of R- and S-Carvedilol in Healthy Japanese Volunteers(Biopharmacy)
OJ-326 The Genotype of CYP2D6 Determines the Cardiovascular Responsiveness to Isoproterenol in Patients Treated with Metoprolol(Cardiovascular Pharmacology, Basic/Clinical 2 (H) : OJ39)(Oral Presentation (Japanese))
OJ-317 Brief Ischemia before Sustained Ischemia Attenuates the Cardiac Sympathetic Nerve Injury via the Reduction of Norepinephrine Release during Ischemia(Nuclear Cardiology 7 (I) : OJ38)(Oral Presentation (Japanese))
Insulin-Stimulated Glucose Transport and Fatty Acid Oxidation Are Impaired in Hypertrophied Hearts before Heart Failure
Effect of Carvedilol on Fatty Acid Metabolism in Rat Hearts with Ischemia-Reperfusion Injury
Normal tracer uptake in early stage after a transient myocardial ischemia does not necessarily imply uninjured fatty acid metabolism
Cardiac responsiveness to β-agonist in a heart after transient ischemia could not be predicted from the cardiac MIBG uptake

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