Nonomura Makoto | The Second Department Of Internal Medicine University Of Toyama School Of Medicine

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著作論文

Supersensitive Response to Isoproterenol in Patients With Marked Global Reduction of Cardiac Metaiodobenzylguanidine Uptake
Cardiac Sympathetic Nerve Activity Alters Myocardial BMIPP Uptake and Its Clearance
Sympathetic Neuronal Function Is Coupled with the Downregulation of β-Adrenoceptors in Ischemia-Reperfusion Rat Hearts
Cardiac washout rate of MIBG is better able to represent sympathetic nerve activity when compared at a comparable MIBG uptake
Early Administration of Fluvastatin, but not at the Onset of Ischemia or Reperfusion, Attenuates Myocardial Ischemia-Reperfusion Injury Through the Nitric Oxide Pathway Rather Than Its Antioxidant Property
Brief Episode of Myocardial Ischemia Before Prolonged Ischemia Attenuates Cardiac Sympathetic Nerve Injury
PJ-780 Hyperglycemia at Reperfusion Dose Not Exacerbate Myocardial Ischemia-reperfusion Injury(Myocardial ischemia-reperfusion, basic/clinical-6 (IHD) PJ136,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulati
PJ-728 A Combined Therapy of AT_1-receptor Antagonist may Enhance the Plaque-stabilizing Effect of Statin(Atherosclerosis, clinical-15 (H) PJ123,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
FRS-036 Beneficial Effects of Ischemic Preconditioning and Diabetes on Myocardial Fatty Acid Metabolism after Ischemia-reperfusion(Advances in Nuclear Cardiology-2 (I) FRS8,Featured Research Session,The 70th Anniversary Annual Scientific Meeting of the Ja
Ischemia-Induced Norepinephrine Release, but not Norepinephrine-Derived Free Radicals, Contributes to Myocardial Ischemia : Reperfusion Injury
Reduced Cardiac Norepinephrine Release during Ischemia is not Necessarily Accompanied by Attenuation of Sympathetic Neuronal Dysfunction in the Remote Period (Nuclear Cardiology 11(I), The 69th Annual Scientific Meeting of the Japanese Circulation Society
The Genotype of CYP2D6 Determines Cardiac Responsiveness to β-adrenergic Stimulation in patients Treated with Metoprolol, but not with Bisoprolol(Cardiovascular Pharmacology, Basic/Clinical 2 (H), The 69th Annual Scientific Meeting of the Japanese Circula
Improvement of Insulin Signaling Induced by Enalapril Contributes to the Inhibition of Developing Hypertensive Heart Failure of Dahl Rats(Nuclear Cardiology 6 (I), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OJ-321 Inhibition of Increasing Synaptic Norepinephrine during Ischemia by Valsartan Protects Cardiac Sympathetic Neuronal Function and β-Receptors from Ischemia-Reperfusion Injury(Nuclear Cardiology 7 (I) : OJ38)(Oral Presentation (Japanese))
OJ-318 Beneficial effects of β-adrenergic blocker in ischemia-reperfusion injury might partially result from the inhibition of non-metabolite accumulation of fatty acid(Nuclear Cardiology 7 (I) : OJ38)(Oral Presentation (Japanese))
Effects of Valsartan on the Ischemia-Reperfusion Injury of Cardiac Sympathetic Nerve Function and β-Adrenoceptors
Insulin-Stimulated Glucose Transport and Fatty Acid Oxidation Are Impaired in Hypertrophied Hearts before Heart Failure
Effect of Carvedilol on Fatty Acid Metabolism in Rat Hearts with Ischemia-Reperfusion Injury
Normal tracer uptake in early stage after a transient myocardial ischemia does not necessarily imply uninjured fatty acid metabolism
Cardiac responsiveness to β-agonist in a heart after transient ischemia could not be predicted from the cardiac MIBG uptake

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