Inoue Minoru | Research Laboratories, Dai-ichi Radioisotope

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Reduced Cardiac Norepinephrine Release during Ischemia is not Necessarily Accompanied by Attenuation of Sympathetic Neuronal Dysfunction in the Remote Period (Nuclear Cardiology 11(I), The 69th Annual Scientific Meeting of the Japanese Circulation Society
Improvement of Insulin Signaling Induced by Enalapril Contributes to the Inhibition of Developing Hypertensive Heart Failure of Dahl Rats(Nuclear Cardiology 6 (I), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
OJ-321 Inhibition of Increasing Synaptic Norepinephrine during Ischemia by Valsartan Protects Cardiac Sympathetic Neuronal Function and β-Receptors from Ischemia-Reperfusion Injury(Nuclear Cardiology 7 (I) : OJ38)(Oral Presentation (Japanese))
OJ-318 Beneficial effects of β-adrenergic blocker in ischemia-reperfusion injury might partially result from the inhibition of non-metabolite accumulation of fatty acid(Nuclear Cardiology 7 (I) : OJ38)(Oral Presentation (Japanese))
Effects of Valsartan on the Ischemia-Reperfusion Injury of Cardiac Sympathetic Nerve Function and β-Adrenoceptors
OJ-317 Brief Ischemia before Sustained Ischemia Attenuates the Cardiac Sympathetic Nerve Injury via the Reduction of Norepinephrine Release during Ischemia(Nuclear Cardiology 7 (I) : OJ38)(Oral Presentation (Japanese))
Cardiac responsiveness to β-agonist in a heart after transient ischemia could not be predicted from the cardiac MIBG uptake

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