PE-222 Alteration of Akt-GSK-3β Signaling by ER Stress Underlies Failure of Erythropoietin to Protect Diabetic Hearts(Myocardial ischemia/reperfusion, basic/clinical(03)(IHD),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circu

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概要

• 論文の詳細を見る
• 社団法人日本循環器学会の論文
• 2008-03-01

著者

• Shimamoto Kazuaki

  Second Department Of Medicine Sapporo Medical University School Of Medicine

• MIURA Tetsuji

  Second Department of Internal Medicine, Sapporo Medical University School of Medicine

• Satoh Takahiro

  Second Department Of Internal Medicine Sapporo Medical University School Of Medicine

• Shimamoto Kazuaki

  Second Department Of Internal Medicine Sapporo Medical University

• Miki Takayuki

  Second Department of Internal Medicine, Sapporo Medical University

• Tanno Masaya

  Second Department of Internal Medicine, Sapporo Medical University School of Medicine

• Yano Toshiyuki

  Second Department of Internal medicine, Sapporo Medical University

• Tanno Masaya

  Second Department Of Medicine Sapporo Medical University School Of Medicine

• Miki Takayuki

  Second Department Of Internal Medicine Sapporo Medical University School Of Medicine

• Miura Tetsuji

  Second Department Of Internal Medicine Sapporo Medical University School Of Medicine

• Miura Tetsuji

  Second Department Of Internal Medicine Sapporo Medical University

• Miura Tetsuji

  The Second Department Of Internal Medicine Sapporo Medical College

• Miura Tetsuji

  Second Department Of Internal Medicine Department Of Clinical Laboratory Medicine Sapporo Medical Un

• Terashima Yoshiaki

  Second Department of Internal Medicine, Sapporo Medical University School of Medicine

• Hotta Hiroyuki

  Second Department of Internal Medicine, Sapporo Medical University School of Medicine

• Simamoto Kazuaki

  Second Department of Internal Medicine, Sapporo Medical University School of Medicine

• Terashima Yoshiaki

  Second Department Of Medicine Sapporo Medical University School Of Medicine

• Miura Tetsuji

  Second Department Of Medicine Sapporo Medical University School Of Medicine

• Hotta Hiroyuki

  Second Department Of Internal Medicine Sapporo Medical University School Of Medicine

• Yano Toshiyuki

  Second Department Of Medicine Sapporo Medical University School Of Medicine

• Miki Takayuki

  Second Department Of Medicine Sapporo Medical University School Of Medicine

関連論文

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• Visceral Obesity in Japanese Patients with Metabolic Syndrome : Reappraisal of Diagnostic Criteria by CT Scan
• Noninvasive Assessment of Left Atrial Function by Strain Rate Imaging in Patients with Hypertension : A Possible Beneficial Effect of Renin-Angiotensin System Inhibition on Left Atrial Function
• Persistent Insulin-Sensitizing Effects of Sarpogrelate Hydrochloride, a Serotonin 2A Receptor Antagonist, in Patients With Peripheral Arterial Disease
• Long-Term Outcome of Implanted Cardioverter Defibrillators in Survivors of Out-of-Hospital Cardiac Arrest of Cardiac Origin
• Role of Adiponectin in Insulin-Resistant Hypertension and Atherosclerosis
• Chinese Medicine, Jiang-Tang-Ke-Li, Improves Insulin Resistance by Modulating Muscle Fiber Composition and Muscle Tumor Necrosis Factor-α in Fructose-Fed Rate
• IS073 Infarct Size-limiting Effect of Ischemic Preconditioning is Disturbed by Post-infarct Ventricular Remodeling via Angiotensin II Type 1 Receptor Activation
• IS071 Infarct Size Limitation by Sodium-hydrogen Exhange Inhibition Requires Activation of Mitochondrial ATP-senstive K Channeles
• IS065 ATP-sensitive potassium channels may play a permissive role in cardioprotection by the Na^+-H^+ exchange inhibitor HOE642 against ischemia/reperfusion injury
• INHIBITION OF THROMBOXANE A2 SYNTHETASE FAILED TO LIMIT MYOCARDIAL INFARCT SIZE IN A RABBIT ISCHEMIA-REPERFUSION MODEL
• Endothelium-dependent Coronary Response is Impaired in the Myocardium at an Early Phase of Post-infarct Remodeling
• Development and Progression of Atherosclerotic Disease in Relation to Insulin Resistance and Hyperinsulinemia
• Hepatocyte Growth Factor and 24-Hour Ambulatory Blood Pressure Monitoring
• OJ-104 Connexin-43 Contributes to Pro-survival Akt/GSK-3β signaling And Cardimyocyte Protection in a Receptor-specifit Manner(Cell death, apoptosis/necrosis(M),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Soci
• OJ-103 Role of Intra-mitochondrial GSK-3β in Bax Regulation And Antiapoptotic Resistance of Cardiomyocytes(Cell death, apoptosis/necrosis(M),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
• PE-268 Ischemic Preconditioning Modifies Interactions between Connexin43 and Protein Kinases during Myocardial Ischemia(Myocardial ischemia-reperfusion, basic/clinical-4 (IHD) PE45,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of
• OE-080 Role of GSK-3 β as a Crucial Determinant of Anti-infarct Tolerance of the Myocardium(Acute myocardial infarction, basic-1 (IHD) OE14,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
• OE-026 Activation of ERK and Suppression of Calcineurin are Interacting Mechanisms of Cardioprotection Afforded by δ-opioid Receptor Activation(Myocardial ischemia-reperfusion, basic/clinical-1 (IHD) OE5,Oral Presentation (English),The 70th Anniversary An
• Activation of δ-Opioid Receptor Suppresses Myocardial Ischemic Injury by ERK Activation during the Early Phase of Ischemia(Acute Myocardial Infarction, Basic 1 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Activation of the δ-opioid Receptor Induces Phosphorylation of Connexin43 at Ser368 by PKC-ε and Reduces Myocardial Infarct Size(Myocardial Ischemia-reperfusion, Basic 3 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Erythropoietin Reduces Myocardial Ischemic Injury, but not Reperfusion Injury, Through Activation of the Pl3k/Akt Pathway(Myocardial Ischemia-reperfusion, Basic 1 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• PE-069 Suppression of Border Zone Remodeling by G-CSF/M-CSF Treatment Improves Ventricular Function after Infarction(Acute Myocardial Infarction, Basic 3 (IHD) : PE12)(Poster Session (English))
• OE-080 Shedding of TNF-a by Activation of ADAM17 Contributes to Activation of Tyrosine Kinase-Mediated Pathway in Ischemic Preconditioning(Myocardial Ischemia-Reperfusion, Basic and Clinical 1 (IHD) : OE10)(Oral Presentation (English))
• OE-079 Suppression of Gap Junction Permeability Is a Mechanism of Cardioprotection Afforded by Activation of the Mitochondrial KATP Channel(Myocardial Ischemia-Reperfusion, Basic and Clinical 1 (IHD) : OE10)(Oral Presentation (English))
• OE-003 Mechanisms of Improvement in Post-infarct Ventricular Function by M-CSF in the rat : Increased Myocyte Regeneration vs. Modulation of Infarct Repair(Acute Myocardial Infarction, Basic 1 (IHD) : OE1)(Oral Presentation (English))
• FRS-084 Ischemia/Reperfusion Up-regulates Egr-1 Expression by an NO and ERK-mediated Mechanism in the Heart.(NO and Cardiovascular System (H) : FRS10)(Featured Research Session (English))
• Microtubules Play a Major Role in Signal Transduction in the Mechanism of Cardioprotection Afforded by Ischemic Preconditioning
• Metaloprotease-Disintegrin Is Involved in Tyrosine Kinase-Mediated Signaling in Ischemic Preconditioning
• PE-545 Distinct Roles of FOXO Subtypes in SIRT1-mediated Mn-SOD Up-regulation under Oxidative Stress(PE091,Molecular Biology (M),Poster Session (English),The 73rd Annual Scientific Meeting of the Japanese Circulation Society)
• DPE-012 Activation of SIRT1, a Histone/Protein Deacetylase, by Resveratrol Up-regulates MnSOD and Suppresses the Progression of Heart Failure(DPE02,Heart Failure, Basic (M),Digital Poster Session (English),The 73rd Annual Scientific Meeting of The Japanes
• Gap Junction Is an End-Effector of Cardioprotection in Ischemic Preconditioning
• Contribution of gap junction communication to myocardial necrosis during ischemia/reperfusion
• Early Defibrillation and Circulatory Support Can Provide Better Long-Term Outcomes Through Favorable Neurological Recovery in Patients With Out-of-Hospital Cardiac Arrest of Cardiac Origin
• OE-036 Roles of GSK-3 β in Myocardial Protection Afforded by Activation of the Mitochondrial K_ Channel(OE06,Myocardial Ischemia-reperfusion (Basic) (IHD),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Soci
• OE-010 Distinct Roles of PKC ε and p38MAPK in Preconditioning-induced Modulation of Gap Junction Permeability during Ischemia(OE02,ACS/AMI (Basic) (IHD),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
• OE-008 Homodimeric Erythropoietin (EPO) Receptor Provokes More Potent and Distinct Cytoprotective Signaling Compared with Heterodimeric EPO-common-β Subunit Receptor in the Heart(OE02,ACS/AMI (Basic) (IHD),Oral Presentation (English),The 73rd Annual Scien
• FRS-044 ROS-mediated Enhancement of ANT-cyclophilin-D Interaction Underlies Reduced Anti-infarct Tolerance and Lost Response of Cardiomyocytes to Pro-survival Signaling in Hypertrophied Hearts(FRS9,Novel Mechanisms of Myocardial Ischemia (IHD),Featured Re
• PJ-005 Impairment of Myocardial Response to Erythropoietin-Induced Protection in Hypertensive Hypertrophied Hearts(Acute myocardial infarction, basic(02)(IHD),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
• PE-333 Resveratrol, An Activator of Longevity Protein SIRT1, Suppresses Progression of Heart Failure Through SIRT1-mediated Cardiomyocyte Protection Against Oxidant Stress(Heart failure, basic(03)(M),Poster Session(English),The 72nd Annual Scientific Meet
• PE-222 Alteration of Akt-GSK-3β Signaling by ER Stress Underlies Failure of Erythropoietin to Protect Diabetic Hearts(Myocardial ischemia/reperfusion, basic/clinical(03)(IHD),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circu
• OE-309 Relationship of the Mitochondrial K_ Channel and GSK-3β in Enhancement of Myocardial Tolerance against Infarction(Myocardial ischemia/reperfusion, basic/clinical(01)(IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Jap
• PE-443 Role of Glycogen Synthase Kinase-3 β in Cardiomyocyte Protection by Erythropoietin(Heart failure, basic-5, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• PE-072 Ischemic Preconditioning Prevents Ischemia/reperfusion-Induced Dephosphorylation of Mitochondrial Connexin-43 in a Protein Complex with Adenine Nucleotide Translocator(Myocardial ischemia/reperfusion, basic/clinical-2, The 71st Annual Scientific Me
• PE-067 Relationships between p38MAPK and PKC in Connexin43-mediated Gap Junction Modulation by Ischemic Preconditioning(Myocardial ischemia/reperfusion, basic/clinical-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• OE-312 Nuclear Localization of SIRT1, a Novel Mechanism of Cardiomyocyte Protection in Failing Hearts, is Achieved by PI3K/Akt Signaling(Heart failure, basic-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• OE-126 Modulation of Mitochondrial Permeability Transition Pore Complex in GSK-3β-mediated Cardiomyocyte Protection(Myocardial ischemia/reperfusion, basic/clinical-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• OE-053 Cardioprotective Signaling Provoked by Erythropoietin is Impaired in Diabetic Myocardium(Acute myocardial infarction, basic-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
• PJ-046 Predictors of Improvement of Exercise Capacity by Nitrates in Experimental Heart Failure(Heart failure, basic-7 (M) PJ8,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
• PE-601 Molecular Mechanisms of Nucleo-cytoplasmic Shuttling of SIRT1, a Histone Deacetylase, and Its Pathopysicological Roles in Cardiomyocytes(Molecular biology, myocardium-2 (M) PE106,Poster Session (English),The 70th Anniversary Annual Scientific Meeti
• Impact of Abnormal Glucose Tolerance, Hypertension and Other Risk Factors on Coronary Artery Disease
• THE EFFECT OF INOTROPIC DOSE OF DOBUTAMINE DURING REPERFUSION ON MYOCARDIAL INFARCT SIZE
• Decreased Safety Factor in the Low Voltage Zone Evoked by Pilsicainide is Critical for Spontaneous Termination of Atrial Reentrant Tachycardia(Arrhythmia, Pharmacological Therapy 1 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Societ
• Modulated Low Voltage Zone Can Provide Dynamic Conversion of Unmappable Atrial Tachycardia by Enhancing a Safety Factor(Arrhythmia, Diagnosis/Pathophysiology/EPS 10 (A), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• PJ-010 Dynamic Conversion of Atrial Flutter by Pilsicainide Studied by a Noncontact 3D Mapping in the Canine Right Atrial Incision Model(Arrhythmia, Diagnosis/Pathophysiology/EPS 4 (A): PJ2)(Poster Session (Japanese))
• HUMAN SUPEROXIDE DISMUTASE FAILED TO LIMIT THE SIZE OF MYOCARDIAL INFARCT AFTER 20-, 30-, OR 60-MINUTE ISCHEMIA AND 72-HOUR REPERFUSION IN THE RABBIT
• Are Treatment Effects of ACEI and ARB in Post-MI Patients Homogeneous?
• Association of Insulin Resistance and Hyperinsulinemia with Disturbed Lipid Metabolism in Patients with Essential Hypertension
• PE-115 Activation of ERK Compensates Disrupted PI3K-Akt Signaling in EPO-induced Cardioprotection of Post-infarct Remodeled Hearts(Acute myocardial infarction, basic-3 (IHD) PE20,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of t
• OE-025 Cardioprotection by Erythropoietin is Associated with Translocation of Akt to Mitochondria and Its Interaction with the Adenine Nucleotide Translocase(Myocardial ischemia-reperfusion, basic/clinical-1 (IHD) OE5,Oral Presentation (English),The 70th
• Protection of Post-infarct Remodeled Myocardium by Erythropoietin is Induced by a PI3K/Akt-independent and Mitochondrial K_ Channel-dependent Mechanism(Myocardial Ischemia-reperfusion, Basic 2 (IHD), The 69th Annual Scientific Meeting of the Japanese
• Erythropoietin Affords Additional Infarct Size-limiting Effects to Preconditioning through Activation of PI3K-Akt-GSK3β Pathway(Acute Myocardial Infarction, Basic 2 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
• Mobilization of Bone Marrow Cells Improves Ventricular Function after Myocardial Infarction by Favorable Effects on Repair in the Infarct-Noninfarct Border
• Ischemic Preconditioning (PC) Induces Dp-Regulation of GLUT4 in the Myocardium : A Possible Mechanism of Late PC against Myocardial Stunning
• Ischemic Preconditioning Enhances Engraftment of Bone Marrow-Derived Stem Cells in the Reperfused Myocardium
• Cytoskeletal Proteins : Hidden Targets of Cytoprotective Signaling?
• Drug Development Targeting the Glycogen Synthase Kinase-3β (GSK-3β)-Mediated Signal Transduction Pathway : Role of GSK-3β in Myocardial Protection Against Ischemia/Reperfusion Injury
• Prognostic implications of left atrial dilation evaluated by echocardiographic volume in patients with normal left atrial antero-posterior dimensions
• The Contribution of Nitric Oxide to Diuretic and Natriuretic Effects of Renal Kinins in Normotensive Rats
• Cloning and Characterization of Homolog of Sir2, a Regulator of Yeast Life Span and Senescence, in the Mouse
• Experimental StudiesEffect of Anoxic Preperfusion on Ischemic Myocardial Injury in Isolated Rat Hearts
• Angiotensin II receptor activation in youth triggers persistent insulin resistance and hypertension-a legacy effect?
• High-Frequent and Spontaneously Diminishing Noises after Shock: A Case of Rare Trouble in Replacement of Implantable Cardioverter-Defibrillator
• Alternations of Transient Outward K+ Current (Ito) in Type 2 Diabetic Ventricular Cardiomyocytes
• Deterioration of Renal Function by Chronic Heart Failure is Associated with Congestion and Oxidative Stress in the Tubulointerstitium
• Three-dimensional transesophageal echocardiography in the diagnosis of atrial septal aneurysm with patent foramen ovale in a patient with ventricular septal defect
• Impact of ICD Shock on Prognosis and Predictors of the Shock
• Bartter's Syndrome with Gouty Arthritis.
• Progression of myocardial infarction in a collateral flow deficient species.

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