Miki Takayuki | Second Department of Internal Medicine, Sapporo Medical University

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著作論文

IS071 Infarct Size Limitation by Sodium-hydrogen Exhange Inhibition Requires Activation of Mitochondrial ATP-senstive K Channeles
OJ-104 Connexin-43 Contributes to Pro-survival Akt/GSK-3β signaling And Cardimyocyte Protection in a Receptor-specifit Manner(Cell death, apoptosis/necrosis(M),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Soci
OJ-103 Role of Intra-mitochondrial GSK-3β in Bax Regulation And Antiapoptotic Resistance of Cardiomyocytes(Cell death, apoptosis/necrosis(M),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PE-268 Ischemic Preconditioning Modifies Interactions between Connexin43 and Protein Kinases during Myocardial Ischemia(Myocardial ischemia-reperfusion, basic/clinical-4 (IHD) PE45,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of
OE-080 Role of GSK-3 β as a Crucial Determinant of Anti-infarct Tolerance of the Myocardium(Acute myocardial infarction, basic-1 (IHD) OE14,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
Activation of δ-Opioid Receptor Suppresses Myocardial Ischemic Injury by ERK Activation during the Early Phase of Ischemia(Acute Myocardial Infarction, Basic 1 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Activation of the δ-opioid Receptor Induces Phosphorylation of Connexin43 at Ser368 by PKC-ε and Reduces Myocardial Infarct Size(Myocardial Ischemia-reperfusion, Basic 3 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Erythropoietin Reduces Myocardial Ischemic Injury, but not Reperfusion Injury, Through Activation of the Pl3k/Akt Pathway(Myocardial Ischemia-reperfusion, Basic 1 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PE-069 Suppression of Border Zone Remodeling by G-CSF/M-CSF Treatment Improves Ventricular Function after Infarction(Acute Myocardial Infarction, Basic 3 (IHD) : PE12)(Poster Session (English))
OE-080 Shedding of TNF-a by Activation of ADAM17 Contributes to Activation of Tyrosine Kinase-Mediated Pathway in Ischemic Preconditioning(Myocardial Ischemia-Reperfusion, Basic and Clinical 1 (IHD) : OE10)(Oral Presentation (English))
OE-079 Suppression of Gap Junction Permeability Is a Mechanism of Cardioprotection Afforded by Activation of the Mitochondrial KATP Channel(Myocardial Ischemia-Reperfusion, Basic and Clinical 1 (IHD) : OE10)(Oral Presentation (English))
OE-003 Mechanisms of Improvement in Post-infarct Ventricular Function by M-CSF in the rat : Increased Myocyte Regeneration vs. Modulation of Infarct Repair(Acute Myocardial Infarction, Basic 1 (IHD) : OE1)(Oral Presentation (English))
FRS-084 Ischemia/Reperfusion Up-regulates Egr-1 Expression by an NO and ERK-mediated Mechanism in the Heart.(NO and Cardiovascular System (H) : FRS10)(Featured Research Session (English))
Metaloprotease-Disintegrin Is Involved in Tyrosine Kinase-Mediated Signaling in Ischemic Preconditioning
DPE-012 Activation of SIRT1, a Histone/Protein Deacetylase, by Resveratrol Up-regulates MnSOD and Suppresses the Progression of Heart Failure(DPE02,Heart Failure, Basic (M),Digital Poster Session (English),The 73rd Annual Scientific Meeting of The Japanes
Contribution of gap junction communication to myocardial necrosis during ischemia/reperfusion
OE-036 Roles of GSK-3 β in Myocardial Protection Afforded by Activation of the Mitochondrial K_ Channel(OE06,Myocardial Ischemia-reperfusion (Basic) (IHD),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Soci
OE-010 Distinct Roles of PKC ε and p38MAPK in Preconditioning-induced Modulation of Gap Junction Permeability during Ischemia(OE02,ACS/AMI (Basic) (IHD),Oral Presentation (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
OE-008 Homodimeric Erythropoietin (EPO) Receptor Provokes More Potent and Distinct Cytoprotective Signaling Compared with Heterodimeric EPO-common-β Subunit Receptor in the Heart(OE02,ACS/AMI (Basic) (IHD),Oral Presentation (English),The 73rd Annual Scien
FRS-044 ROS-mediated Enhancement of ANT-cyclophilin-D Interaction Underlies Reduced Anti-infarct Tolerance and Lost Response of Cardiomyocytes to Pro-survival Signaling in Hypertrophied Hearts(FRS9,Novel Mechanisms of Myocardial Ischemia (IHD),Featured Re
PJ-005 Impairment of Myocardial Response to Erythropoietin-Induced Protection in Hypertensive Hypertrophied Hearts(Acute myocardial infarction, basic(02)(IHD),Poster Session(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PE-333 Resveratrol, An Activator of Longevity Protein SIRT1, Suppresses Progression of Heart Failure Through SIRT1-mediated Cardiomyocyte Protection Against Oxidant Stress(Heart failure, basic(03)(M),Poster Session(English),The 72nd Annual Scientific Meet
PE-222 Alteration of Akt-GSK-3β Signaling by ER Stress Underlies Failure of Erythropoietin to Protect Diabetic Hearts(Myocardial ischemia/reperfusion, basic/clinical(03)(IHD),Poster Session(English),The 72nd Annual Scientific Meeting of the Japanese Circu
OE-309 Relationship of the Mitochondrial K_ Channel and GSK-3β in Enhancement of Myocardial Tolerance against Infarction(Myocardial ischemia/reperfusion, basic/clinical(01)(IHD),Oral Presentation(English),The 72nd Annual Scientific Meeting of the Jap
PE-443 Role of Glycogen Synthase Kinase-3 β in Cardiomyocyte Protection by Erythropoietin(Heart failure, basic-5, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PE-072 Ischemic Preconditioning Prevents Ischemia/reperfusion-Induced Dephosphorylation of Mitochondrial Connexin-43 in a Protein Complex with Adenine Nucleotide Translocator(Myocardial ischemia/reperfusion, basic/clinical-2, The 71st Annual Scientific Me
PE-067 Relationships between p38MAPK and PKC in Connexin43-mediated Gap Junction Modulation by Ischemic Preconditioning(Myocardial ischemia/reperfusion, basic/clinical-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-126 Modulation of Mitochondrial Permeability Transition Pore Complex in GSK-3β-mediated Cardiomyocyte Protection(Myocardial ischemia/reperfusion, basic/clinical-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-053 Cardioprotective Signaling Provoked by Erythropoietin is Impaired in Diabetic Myocardium(Acute myocardial infarction, basic-1, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PJ-046 Predictors of Improvement of Exercise Capacity by Nitrates in Experimental Heart Failure(Heart failure, basic-7 (M) PJ8,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
PE-115 Activation of ERK Compensates Disrupted PI3K-Akt Signaling in EPO-induced Cardioprotection of Post-infarct Remodeled Hearts(Acute myocardial infarction, basic-3 (IHD) PE20,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of t
OE-025 Cardioprotection by Erythropoietin is Associated with Translocation of Akt to Mitochondria and Its Interaction with the Adenine Nucleotide Translocase(Myocardial ischemia-reperfusion, basic/clinical-1 (IHD) OE5,Oral Presentation (English),The 70th
Protection of Post-infarct Remodeled Myocardium by Erythropoietin is Induced by a PI3K/Akt-independent and Mitochondrial K_ Channel-dependent Mechanism(Myocardial Ischemia-reperfusion, Basic 2 (IHD), The 69th Annual Scientific Meeting of the Japanese
Erythropoietin Affords Additional Infarct Size-limiting Effects to Preconditioning through Activation of PI3K-Akt-GSK3β Pathway(Acute Myocardial Infarction, Basic 2 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Mobilization of Bone Marrow Cells Improves Ventricular Function after Myocardial Infarction by Favorable Effects on Repair in the Infarct-Noninfarct Border
Ischemic Preconditioning (PC) Induces Dp-Regulation of GLUT4 in the Myocardium : A Possible Mechanism of Late PC against Myocardial Stunning
Ischemic Preconditioning Enhances Engraftment of Bone Marrow-Derived Stem Cells in the Reperfused Myocardium
Drug Development Targeting the Glycogen Synthase Kinase-3β (GSK-3β)-Mediated Signal Transduction Pathway : Role of GSK-3β in Myocardial Protection Against Ischemia/Reperfusion Injury

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