Ikeda Yoshihiro | Second Department Of Internal Medicine Sapporo Medical University

スポンサーリンク

概要

関連著者

もっと見る

著作論文

OJ-104 Connexin-43 Contributes to Pro-survival Akt/GSK-3β signaling And Cardimyocyte Protection in a Receptor-specifit Manner(Cell death, apoptosis/necrosis(M),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Soci
OJ-103 Role of Intra-mitochondrial GSK-3β in Bax Regulation And Antiapoptotic Resistance of Cardiomyocytes(Cell death, apoptosis/necrosis(M),Oral Presentation(Japanese),The 72nd Annual Scientific Meeting of the Japanese Circulation Society)
PE-268 Ischemic Preconditioning Modifies Interactions between Connexin43 and Protein Kinases during Myocardial Ischemia(Myocardial ischemia-reperfusion, basic/clinical-4 (IHD) PE45,Poster Session (English),The 70th Anniversary Annual Scientific Meeting of
OE-080 Role of GSK-3 β as a Crucial Determinant of Anti-infarct Tolerance of the Myocardium(Acute myocardial infarction, basic-1 (IHD) OE14,Oral Presentation (English),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation Society)
OE-026 Activation of ERK and Suppression of Calcineurin are Interacting Mechanisms of Cardioprotection Afforded by δ-opioid Receptor Activation(Myocardial ischemia-reperfusion, basic/clinical-1 (IHD) OE5,Oral Presentation (English),The 70th Anniversary An
Activation of δ-Opioid Receptor Suppresses Myocardial Ischemic Injury by ERK Activation during the Early Phase of Ischemia(Acute Myocardial Infarction, Basic 1 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Activation of the δ-opioid Receptor Induces Phosphorylation of Connexin43 at Ser368 by PKC-ε and Reduces Myocardial Infarct Size(Myocardial Ischemia-reperfusion, Basic 3 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Erythropoietin Reduces Myocardial Ischemic Injury, but not Reperfusion Injury, Through Activation of the Pl3k/Akt Pathway(Myocardial Ischemia-reperfusion, Basic 1 (IHD), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PE-069 Suppression of Border Zone Remodeling by G-CSF/M-CSF Treatment Improves Ventricular Function after Infarction(Acute Myocardial Infarction, Basic 3 (IHD) : PE12)(Poster Session (English))
OE-080 Shedding of TNF-a by Activation of ADAM17 Contributes to Activation of Tyrosine Kinase-Mediated Pathway in Ischemic Preconditioning(Myocardial Ischemia-Reperfusion, Basic and Clinical 1 (IHD) : OE10)(Oral Presentation (English))
OE-079 Suppression of Gap Junction Permeability Is a Mechanism of Cardioprotection Afforded by Activation of the Mitochondrial KATP Channel(Myocardial Ischemia-Reperfusion, Basic and Clinical 1 (IHD) : OE10)(Oral Presentation (English))
OE-003 Mechanisms of Improvement in Post-infarct Ventricular Function by M-CSF in the rat : Increased Myocyte Regeneration vs. Modulation of Infarct Repair(Acute Myocardial Infarction, Basic 1 (IHD) : OE1)(Oral Presentation (English))
FRS-084 Ischemia/Reperfusion Up-regulates Egr-1 Expression by an NO and ERK-mediated Mechanism in the Heart.(NO and Cardiovascular System (H) : FRS10)(Featured Research Session (English))
Microtubules Play a Major Role in Signal Transduction in the Mechanism of Cardioprotection Afforded by Ischemic Preconditioning
Metaloprotease-Disintegrin Is Involved in Tyrosine Kinase-Mediated Signaling in Ischemic Preconditioning

もっと見る

スポンサーリンク