Ono Hideki | Laboratory Of Cns Pharmacology Graduate School Of Pharmaceutical Sciences

スポンサーリンク

概要

関連著者

もっと見る

著作論文

Taltirelin Improves Motor Ataxia Independently of Monoamine Levels in Rolling Mouse Nagoya, a Model of Spinocerebellar Atrophy(Pharmacology)
Enhanced Wind-Up of the C-Fiber-Mediated Nociceptive Flexor Reflex Movement Following Painful Diabetic Neuropathy in Mice
Noxious Stimuli Evoke a Biphasic Flexor Reflex Composed of Aδ-Fiber-Mediated Short-Latency and C-Fiber-Mediated Long-Latency Withdrawal Movements in Mice
Oseltamivir, an Anti-influenza Virus Drug, Produces Hypothermia in Mice : Comparison Among Oseltamivir, Zanamivir and Diclofenac(Pharmacology)
Spinorphin, an Endogenous Inhibitor of Enkephalin-Degrading Enzymes, Potentiates Leu-Enkephalin-Induced Anti-allodynic and Antinociceptive Effects in Mice
Protein Kinase A-Dependence of the Supraspinally Mediated Analgesic Effects of Gabapentin on Thermal and Mechanical Hypersensitivity
The Supraspinally Mediated Analgesic Effects of Zonisamide in Mice After Peripheral Nerve Injury Are Independent of the Descending Monoaminergic System
Glycine Transporter Blockade Ameliorates Motor Ataxia in a Mouse Model of Spinocerebellar Atrophy
Involvement of Supraspinal Imidazoline Receptors and Descending Monoaminergic Pathways in Tizanidine-Induced Inhibition of Rat Spinal Reflexes
GABA_B Receptors Do Not Mediate the Inhibitory Actions of Gabapentin on the Spinal Reflex in Rats
Antinociceptive Effects of Sodium Channel-Blocking Agents on Acute Pain in Mice
Endogenous GABA Does Not Mediate the Inhibitory Effects of Gabapentin on Spinal Reflexes in Rats
Neuroprotection via Strychnine-Sensitive Glycine Receptors During Post-ischemic Recovery of Excitatory Synaptic Transmission in the Hippocampus
Zonisamide Suppresses Pain Symptoms of Formalin-Induced Inflammatory and Streptozotocin-Induced Diabetic Neuropathy
Neuroprotection via Strychnine-Sensitive Glycine Receptors During Post-ischemic Recovery of Excitatory Synaptic Transmission in the Hippocampus
Method for Recording Spinal Reflexes in Mice: Effects of Thyrotropin-Releasing Hormone, DOI, Tolperisone and Baclofen on Monosynaptic Spinal Reflex Potentials
N- and L-Type Voltage-Dependent Ca^ Channels Contribute to the Generation of After-Discharges in the Spinal Ventral Root After Cessation of Noxious Mechanical Stimulation
Effect of Spinally Administered Simvastatin on the Formalin-Induced Nociceptive Response in Mice
Effect of Spinally Administered Simvastatin on the Formalin-Induced Nociceptive Response in Mice
Antinociceptive Effects of Sodium Channel-Blocking Agents on Acute Pain in Mice
Contribution of TRPV1 Receptor-Expressing Fibers to Spinal Ventral Root After-Discharges and Mechanical Hyperalgesia in a Spared Nerve Injury (SNI) Rat Model

もっと見る

スポンサーリンク