Arai Masazumi | Department Of Cardiology Gifu University Graduate School Of Medicine

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著作論文

Granulocyte Colony-Stimulating Factor : A Noninvasive Regeneration Therapy for Treating Atherosclerotic Peripheral Artery Disease
OJ-254 Erythropoietin Drug Delivery System protects the heart against myocardial infarction through activation of Akt and angiogenesis(Growth factor/Cytokine-2(H/M), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-128 Pioglitazone, an Anti-Diabetic Drug, Reduces Myocardial Infarct Size via Stimulating Peroxisome Proliferator Activated Receptor-γ (PPARγ) in Rabbits Model of Myocardial Infarction(Myocardial ischemia/reperfusion, basic/clinical-1, The 71st Annual S
PJ-571 Multiple Mechanisms underlie Long QT Syndrome by the Change of Amino Acid R259 in the Intracellular S4-S5 Linker of KCNQ1(PJ096,Arrhythmia, Others (Clinical/Pathophysiology) 4 (A),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The
PE-141 Clopidogrel Resistance in Japanese Patients Scheduled for Percutaneous Coronary Intervention(PE024,Coronary Revascularization, PCI (Restenosis/Others) 1 (IHD),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation S
PE-106 SCN5A and Lamin A/C Gene Mutations are Highly Prevalent in Patients with Familial Bradyarrhythmic Disorders(PE018,Arrhythmia, Others (Clinical/Pathophysiology) (A),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulat
OJ-314 Drug Delivery System Using Erythropoietin Gelatin Hydrogel Sheet Decreases Myocardial Infarct Size and Improves Left Ventricular Function and Remodeling(Growth factor/Cytokine-1 (H) OJ53,Oral Presentation (Japanese),The 70th Anniversary Annual Scie
PJ-340 Curcumin, a Natural p300-specific Histone Acetyltransferase Inhibitor, Possesses Beneficial Effects in addition to ACE Inhibitor after Myocardial Infarction in Rats(PJ057,Translational Science (Cardiac) (H),Poster Session (Japanese),The 73rd Annual
PE-351 Synergistic Effects of G-CSF and Erythropoietin on Left Ventricular Systolic Function in Heart Failure after Rat Myocardial Infarction(PE059,Heart Failure (Treatment) 2 (M),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese
DPJ-032 Improvement of Severe Anemia in Patients with Aortic Stenosis Complicated with Gastrointestinal Angiodysplasia (Heyde's Syndrome) after Aortic Valve Repair(DPJ06,Valvular Heart Disease/Pericarditis/Cardiac Tumor (M),Digital Poster Session (Japanes
OE-031 Visualization of Accumulating Reactive Oxygen Species during Myocardial Ischemia by Real-time Two-photon Imaging of Perfused Rat Hearts(OE06,Myocardial Ischemia-reperfusion (Basic) (IHD),Oral Presentation (English),The 73rd Annual Scientific Meetin
PJ-440 Ginsenoside Rg1 Reduces Myocardial Infarct Size via Activation of Protein Kinase C and Production of Nitric Oxide in Rabbits(Acute myocardial infarction, basic-4 (IHD) PJ74,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of
PJ-343 Post-infarction Treatment with Ginsenoside Rg1 Improves Cardiac Function and Left Ventricular Remodeling Through Enhancement of Intrinsic G-CSF(Cardiovascular pharmacology, basic/clinical-4 (H) PJ58,Poster Session (Japanese),The 70th Anniversary An
PE-078 Pravastatin Reduces Myocardial Infarct Size Through Nitric Oxide Production, Protein Kinase C Activation, Mitochondrial KATP Channel Opening and Decreasing Oxyradicals(Cardiovascular pharmacology, basic/clinical-3 (H) PE13,Poster Session (English),
Acarbose, an α-glucosidase Inhibitor, Reduces Myocardial Infarct Size in Rabbits Involving the Opening of Mitochondrial KATP Channels(Diabetes/Obesity 8 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Accelerated Post-infarct Neovascularization and Tissue Repair by Chemotherapy-combined G-CSF May Be Related to Peripheral CD34^+ Cells Increased via CXCR4-SDF-1 Axis(Apoptosis/Necrosis/Regeneration 1 (M), The 69th Annual Scientific Meeting of the Japanese
HGF Gene Transfer is Protective Against Ischemia-Reperfusion Injury via Enhancing Angiogenesis and Bcl-2 Expression and Decreasing Hydroxyl Radicals(The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PJ-584 Nicorandil Reduces the Production of Hydroxyl Radicals During Ischemia and Reperfusion Through Opening the Mitochondrial KATP Channels in Rabbit Hearts(Cardiovascular Pharmacology, Basic/Clinical 5 (H) : PJ98)(Poster Session (Japanese))
PJ-377 Healing Process Acceleration May be Important as a Mechanism of Improvement of Cardiac Function and Remodeling by Post-infarct G-CSF Treatment(Apoptosis/Necrosis/Regeneration 1 (M) : PJ63)(Poster Session (Japanese))
PE-270 Intravenous Transplantation of Autologous Bone Marrow-Derived Mononuclear Cells Improves Cardiac Function via Downregulation of TGF-beta and TNF-alpha in Ischemia-Reperfusion Hearts.(Transplantation 1 (M) : PE46)(Poster Session (English))
OE-110 G-CSF after Transient Myelosuppression Improves Cardiac Function via More Myocardial Regeneration and Vasculogenesis than G-CSF Alone in Ischemia-Reperfusion Hearts(Heart Failure, Basic 1 (M) : OE13)(Oral Presentation (English))
Tendai-Uyaku Improves Post-Ischemic Left Ventricular Dysfunction through Inhibiting Production of Hydroxy Radicals and Opening the Mitochondrial KATP Channels
Nicorandil Has a Free Radical Scavenging Effect through Opening the Mitochondrial KATP Channels during Ischemia and Reperfusion in Rabbits
Miglitol Reduces Myocardial Infarct Size via Scavenging Hydroxyl Radicals, Enhancing Bel-XL Expression and Reducing Bax Expression in Rabbits
HGF Gene Transfer Reduces Myocardial Infarct Size through Decreasing Hydroxyl Radicals Production and Enhancing Expression of BcI-2 in Rabbits
G-CSF Treatment after Myocardial Infarction Improves Left Ventricular Function and Remodeling Even at 3 Months after Infarction
PE-104 Severer Phenotypes of Long QT Syndrome are Associated with Compound Mutations : A Multicenter Study(PE018,Arrhythmia, Others (Clinical/Pathophysiology) (A),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese Circulation Soci
FRS-052 Identification and Functional Characterization of a Novel Intronic Splicing Mutation in KCNQ1 Associated with Long QT Syndrome(FRS11,New Insights into Basic Mechanisms of Arrhythmia (A),Featured Research Session (English),The 73rd Annual Scientifi
2 Latent Genetic Backgrounds and Molecular Pathogenesis of Drug-induced Long QT Syndrome(Proarrhythmic Risk of Drugs and Their Evaluation,Symposium 7 (SY-07) (I),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
1 A Novel Genetic Marker for Potential Risk of QT Prolongation and Cardiac Sudden Death : KCNE1-D85N Polymorphism(Biomarkers, Genetic Polymorphism, and Risk Estimation,Symposium 1 (SY-01) (H),The 73rd Annual Scientific Meeting of The Japanese Circulation
Mutation Analysis of the Glycerol-3 Phosphate Dehydrogenase-1 Like (GPD1L) Gene in Japanese Patients With Brugada Syndrome
PE-103 Clinical Phenotype of Japanese LQT3 Form of Congenital Long QT Syndrome by Location of Mutation from Japanese Multicenter Registry(PE018,Arrhythmia, Others (Clinical/Pathophysiology) (A),Poster Session (English),The 73rd Annual Scientific Meeting o
FRS-117 Clinical and Electrocardiographic Characteristics of Japanese Probands with Brugada Syndrome with an SCN5A Mutation from Japanese Multicenter Registry(FRS24,Novel Mechanisms of Ventricular Arrhythmia (A),Featured Research Session (English),The 73r
Sustained Release of Prostaglandin E_1 Potentiates the Impaired Therapeutic Angiogenesis by Basic Fibroblast Growth Factor in Diabetic Murine Hindlimb Ischemia
PJ-545 Significance of Chronic Partial Unloading on Improvement in β-Adrenergic Response and Normalization of Gene Expression in Failing Rat Hearts(PJ092,Transplantation/LVAD/Cardiovascular Surgery/CABG (M/IHD),Poster Session (Japanese),The 73rd Annual Sc
Delivery Route in Bone Marrow Cell Transplantation Should be Optimized According to the Etiology of Heart Disease
Modification of Post-Myocardial Infarction Granulocyte-Colony Stimulating Factor Therapy With Myelosuppressives
Improvement of Quality of Life With Nocturnal Oxygen Therapy in Heart Failure Patients With Central Sleep Apnea
OJ-179 Long Term Effect of Granulocyte Colony-Stimulating Factor as a Novel and Noninvasive Regeneration Therapy Against Atherosclerotic Peripheral Artery Disease(Peripheral Circulation/Vascular Disease 1 (H) : OJ21)(Oral Presentation (Japanese))
2 A low dose but long duration post-myocardial infarction treatment with granulocyte colony-stimulating factor shows a beneficial effect without any complications(Plenary Session 2 (PL-2) (IHD) Myocardial Protection Strategy after Reperfusion Therapy in A
Elastic Anomaly of the Ferroelectric Phase Transition of Li_2Ge_7O_ Studied by the Brillouin Scattering
4 Clinical Trials of Cardiovascular Regeneration Therapy Using Granulocyte Colony-Stimulating Factor(Clinical Trials of Regeneration Therapy in Japan, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Effect of Granulocyte Colony-Stimulating Factor Treatment at a Low Dose but for a Long Duration in Patients With Coronary Heart Disease : A Pilot Study
Cyclophosphamide Improves the Function of Post-Infarct Hearts by Reducing Old Infarct Area and Accelerating the Mobilization of CD34^+ Cells
Postinfarction Treatment With Edaravone, a Free Radical Scavenger, Reduces Myocardial Infarct Size, Improves Cardiac Function and Remodeling in Rabbits(Cardiovascular Pharmacology, Basic/Clinical 2 (H), The 69th Annual Scientific Meeting of the Japanese C
Elastic Instability in the Ferroelastic Phase Transition of TlH_2PO_4 Studied by Brillouin Scattering
4 Regeneration Therapy for Heart Failure due to Nonischemic Cardiomyopathy(Symposium 8 (SY8) (M) : Regeneration Medicine of Cardiovascular Diseases)(Special Program)
A Natural p300-Specific Histone Acetyltransferase Inhibitor, Curcumin, in Addition to Angiotensin-Converting Enzyme Inhibitor, Exerts Beneficial Effects on Left Ventricular Systolic Function After Myocardial Infarction in Rats
Predicting Long-Term Mortality After First Coronary Revascularization

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