Arai Masazumi | Department of Cardiology, Regeneration Medicine and Bioethics, Gifu University Graduate School of Me

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著作論文

Granulocyte Colony-Stimulating Factor : A Noninvasive Regeneration Therapy for Treating Atherosclerotic Peripheral Artery Disease
OJ-254 Erythropoietin Drug Delivery System protects the heart against myocardial infarction through activation of Akt and angiogenesis(Growth factor/Cytokine-2(H/M), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
OE-128 Pioglitazone, an Anti-Diabetic Drug, Reduces Myocardial Infarct Size via Stimulating Peroxisome Proliferator Activated Receptor-γ (PPARγ) in Rabbits Model of Myocardial Infarction(Myocardial ischemia/reperfusion, basic/clinical-1, The 71st Annual S
OJ-314 Drug Delivery System Using Erythropoietin Gelatin Hydrogel Sheet Decreases Myocardial Infarct Size and Improves Left Ventricular Function and Remodeling(Growth factor/Cytokine-1 (H) OJ53,Oral Presentation (Japanese),The 70th Anniversary Annual Scie
PJ-440 Ginsenoside Rg1 Reduces Myocardial Infarct Size via Activation of Protein Kinase C and Production of Nitric Oxide in Rabbits(Acute myocardial infarction, basic-4 (IHD) PJ74,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of
PJ-343 Post-infarction Treatment with Ginsenoside Rg1 Improves Cardiac Function and Left Ventricular Remodeling Through Enhancement of Intrinsic G-CSF(Cardiovascular pharmacology, basic/clinical-4 (H) PJ58,Poster Session (Japanese),The 70th Anniversary An
PE-078 Pravastatin Reduces Myocardial Infarct Size Through Nitric Oxide Production, Protein Kinase C Activation, Mitochondrial KATP Channel Opening and Decreasing Oxyradicals(Cardiovascular pharmacology, basic/clinical-3 (H) PE13,Poster Session (English),
Acarbose, an α-glucosidase Inhibitor, Reduces Myocardial Infarct Size in Rabbits Involving the Opening of Mitochondrial KATP Channels(Diabetes/Obesity 8 (H), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
Accelerated Post-infarct Neovascularization and Tissue Repair by Chemotherapy-combined G-CSF May Be Related to Peripheral CD34^+ Cells Increased via CXCR4-SDF-1 Axis(Apoptosis/Necrosis/Regeneration 1 (M), The 69th Annual Scientific Meeting of the Japanese
HGF Gene Transfer is Protective Against Ischemia-Reperfusion Injury via Enhancing Angiogenesis and Bcl-2 Expression and Decreasing Hydroxyl Radicals(The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PJ-584 Nicorandil Reduces the Production of Hydroxyl Radicals During Ischemia and Reperfusion Through Opening the Mitochondrial KATP Channels in Rabbit Hearts(Cardiovascular Pharmacology, Basic/Clinical 5 (H) : PJ98)(Poster Session (Japanese))
PJ-377 Healing Process Acceleration May be Important as a Mechanism of Improvement of Cardiac Function and Remodeling by Post-infarct G-CSF Treatment(Apoptosis/Necrosis/Regeneration 1 (M) : PJ63)(Poster Session (Japanese))
PE-270 Intravenous Transplantation of Autologous Bone Marrow-Derived Mononuclear Cells Improves Cardiac Function via Downregulation of TGF-beta and TNF-alpha in Ischemia-Reperfusion Hearts.(Transplantation 1 (M) : PE46)(Poster Session (English))
OE-110 G-CSF after Transient Myelosuppression Improves Cardiac Function via More Myocardial Regeneration and Vasculogenesis than G-CSF Alone in Ischemia-Reperfusion Hearts(Heart Failure, Basic 1 (M) : OE13)(Oral Presentation (English))
Tendai-Uyaku Improves Post-Ischemic Left Ventricular Dysfunction through Inhibiting Production of Hydroxy Radicals and Opening the Mitochondrial KATP Channels
Nicorandil Has a Free Radical Scavenging Effect through Opening the Mitochondrial KATP Channels during Ischemia and Reperfusion in Rabbits
Miglitol Reduces Myocardial Infarct Size via Scavenging Hydroxyl Radicals, Enhancing Bel-XL Expression and Reducing Bax Expression in Rabbits
HGF Gene Transfer Reduces Myocardial Infarct Size through Decreasing Hydroxyl Radicals Production and Enhancing Expression of BcI-2 in Rabbits
G-CSF Treatment after Myocardial Infarction Improves Left Ventricular Function and Remodeling Even at 3 Months after Infarction
Modification of Post-Myocardial Infarction Granulocyte-Colony Stimulating Factor Therapy With Myelosuppressives
OJ-179 Long Term Effect of Granulocyte Colony-Stimulating Factor as a Novel and Noninvasive Regeneration Therapy Against Atherosclerotic Peripheral Artery Disease(Peripheral Circulation/Vascular Disease 1 (H) : OJ21)(Oral Presentation (Japanese))
2 A low dose but long duration post-myocardial infarction treatment with granulocyte colony-stimulating factor shows a beneficial effect without any complications(Plenary Session 2 (PL-2) (IHD) Myocardial Protection Strategy after Reperfusion Therapy in A
Elastic Anomaly of the Ferroelectric Phase Transition of Li_2Ge_7O_ Studied by the Brillouin Scattering
4 Clinical Trials of Cardiovascular Regeneration Therapy Using Granulocyte Colony-Stimulating Factor(Clinical Trials of Regeneration Therapy in Japan, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
Effect of Granulocyte Colony-Stimulating Factor Treatment at a Low Dose but for a Long Duration in Patients With Coronary Heart Disease : A Pilot Study
Cyclophosphamide Improves the Function of Post-Infarct Hearts by Reducing Old Infarct Area and Accelerating the Mobilization of CD34^+ Cells
Postinfarction Treatment With Edaravone, a Free Radical Scavenger, Reduces Myocardial Infarct Size, Improves Cardiac Function and Remodeling in Rabbits(Cardiovascular Pharmacology, Basic/Clinical 2 (H), The 69th Annual Scientific Meeting of the Japanese C
Elastic Instability in the Ferroelastic Phase Transition of TlH_2PO_4 Studied by Brillouin Scattering

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