Chen Xue-hai | Second Department Of Internal Medicine Gifu University School Of Medicine

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著作論文

Celiprolol, a Selective β_1-Blocker, Reduces the Infarct Size Through Production of Nitric Oxide in a Rabbit Model of Myocardial Infarction
Nicorandil Improves Post-Ischemic Myocardial Dysfunction in Association With Opening the Mitochondrial KATP Channels and Decreasing Hydroxyl Radicals in Isolated Rat Hearts
Cilnidipine, an N+L-Type Dihydropyridine Ca Channel Blocker, Suppresses the Occurrence of Ischemia/Reperfusion Arrhythmia in a Rabbit Model of Myocardial Infarction
HGF Gene Transfer is Protective Against Ischemia-Reperfusion Injury via Enhancing Angiogenesis and Bcl-2 Expression and Decreasing Hydroxyl Radicals(The 69th Annual Scientific Meeting of the Japanese Circulation Society)
PJ-584 Nicorandil Reduces the Production of Hydroxyl Radicals During Ischemia and Reperfusion Through Opening the Mitochondrial KATP Channels in Rabbit Hearts(Cardiovascular Pharmacology, Basic/Clinical 5 (H) : PJ98)(Poster Session (Japanese))
PJ-377 Healing Process Acceleration May be Important as a Mechanism of Improvement of Cardiac Function and Remodeling by Post-infarct G-CSF Treatment(Apoptosis/Necrosis/Regeneration 1 (M) : PJ63)(Poster Session (Japanese))
PE-270 Intravenous Transplantation of Autologous Bone Marrow-Derived Mononuclear Cells Improves Cardiac Function via Downregulation of TGF-beta and TNF-alpha in Ischemia-Reperfusion Hearts.(Transplantation 1 (M) : PE46)(Poster Session (English))
OJ-505 Regeneration-Unrelated Mechanisms Underlying Ameliorating Effect of Bone Marrow Cell Transplantation Therapy in Rabbit Hearts with Cardiomyopathy(Cardiomyopathy, Basic (M) : OJ62)(Oral Presentation (Japanese))
OE-110 G-CSF after Transient Myelosuppression Improves Cardiac Function via More Myocardial Regeneration and Vasculogenesis than G-CSF Alone in Ischemia-Reperfusion Hearts(Heart Failure, Basic 1 (M) : OE13)(Oral Presentation (English))
Tendai-Uyaku Improves Post-Ischemic Left Ventricular Dysfunction through Inhibiting Production of Hydroxy Radicals and Opening the Mitochondrial KATP Channels
Nicorandil Has a Free Radical Scavenging Effect through Opening the Mitochondrial KATP Channels during Ischemia and Reperfusion in Rabbits
Miglitol Reduces Myocardial Infarct Size via Scavenging Hydroxyl Radicals, Enhancing Bel-XL Expression and Reducing Bax Expression in Rabbits
HGF Gene Transfer Reduces Myocardial Infarct Size through Decreasing Hydroxyl Radicals Production and Enhancing Expression of BcI-2 in Rabbits
G-CSF Treatment after Myocardial Infarction Improves Left Ventricular Function and Remodeling Even at 3 Months after Infarction
Elucidation of the Roles of HB-EGF in the Heart by the Overexpression of HB-EGF
Intravenous transplantation of autologuos bone marrow cells after myocardial infarction regenerates cardiomyocytes and improvescardiac function in rabbit hearts
Sheng-Mai-San is Protective Against Post-Ischemic Myocardial Dysfunction in Rats Through Its Opening of the Mitochondrial KATP Channels
Celiprolol, a β 1-blocker, reduces myocardiat infarct size not through mitochondrial KATP channel opening but through nitric oxide production in rabbits
Shen-Mai-San is protective against post-ischemic myocardial dysfunction through scavenging hydroxyl radicals and opening the mitochondrial KATP channels in rats
Protective Effect of a Novel Anti-Diabetic Drug, Miglitol, an α-Glucosidase Inhibitor, Against Anginal Ischemia in a Canine Model
ACE inhibitor quinaprilat reduces myocardial infarct size through production of nitric oxide and opening the mitochondrial KATP channels in rabbits
Benidipine reduces infarct size through producton of nitric oxide and scavenging hydroxyl radicals in a rabbit model of myocardial infarction

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