Kobayashi Shigeki | Department Of Medicine And Clinical Science Yamaguchi University Graduate School Of Medicine

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概要

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著作論文

PJ-573 Dantrolene Prevents Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT) by Correcting Abnormal Ca^ Release through the Ryanodine Receptor(PJ096,Arrhythmia, Others (Clinical/Pathophysiology) 4 (A),Poster Session (Japanese),The 73rd Annu
PJ-430 Domain2114-2149 within Ryanodine Receptor (RyR) Plays a Key Role for Stabilization of RyR2 in Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT)(PJ073,Arrhythmia, Others (Basic) 1 (A),Poster Session (Japanese),The 73rd Annual Scientific M
PJ-375 Carperitide, Atrial Natriuretic Peptide, Improves Hemoglobin Level in Acute Heart Failure(PJ063,Heart Failure (Treatment) 1 (M),Poster Session (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Society)
PJ-068 Abnormally Tight Domain-Domain Interaction at Mutation Site could be a Primary Cause of Catecholaminergic Polymorphic Ventricular Tachycardia(PJ012,Calcium Handling, Cardiac Function (Basic) (M),Poster Session (Japanese),The 73rd Annual Scientific
5 Decreased affinity of calmodulin binding to RyR2 may cause leaky channel in CPVT-associated mutation(Young Investigator's Award for International Students Finalists Lecture (YIA International),The 73rd Annual Scientific Meeting of The Japanese Circulati
3 Correction of RyR2 Defects as a New Therapeutic Strategy against Heart Failure(Novel Therapeutic Strategies against Heart Failure,KSC-JCS Joint Symposium (KSC-JCS Joint Symposium) (I),The 73rd Annual Scientific Meeting of The Japanese Circulation Societ
Role of ryanodine receptor as a Ca^ regulatory center in normal and failing hearts
Defective Ca^ Cycling as a Key Pathogenic Mechanism of Heart Failure(Molecular Mechanisms)
PJ-203 Dantrolene, a therapeutic agent for malignant hyperthermia, inhibits catecholaminergic polymorphic ventricular tachycardia : Insights from a RyR2 R2474S/+ knock-in mouse model(Arrhythmia, basic(05)(A),Poster Session(Japanese),The 72nd Annual Scient
FRS-018 Marked Increase in Spontaneous Ca2+ Sparks In Response To β-stimulation As A Critical Cause Of Catecholaminergic Polymorphic Ventricular Tachycardia(New Insights into Basic Mechanism of Arrhythmia(A),Featured Research Session,The 72nd Annual Scien
FRS-017 Enhanced Sensitivity of the Cardiac Ryanodine Receptor to Activation by Luminal Ca^ as a Primary Cause of CPVT(New Insights into Basic Mechanism of Arrhythmia(A),Featured Research Session,The 72nd Annual Scientific Meeting of the Japanese Circ
PJ-765 Scavenging Free Radicals by Low-dose Carvedilol Prevents Redox-dependent Ca^ Leak via Stabilization of Ryanodine Receptor in Heart Failure(Development and differentiation-2, The 71st Annual Scientific Meeting of the Japanese Circulation Society
PJ-103 Defective Domain-Domain Interaction within Ryanodine Receptor as a Critical Cause of Diastolic Ca^ Spark in Cardiomyocytes(Excitation-contraction coupling/Ion channel, The 71st Annual Scientific Meeting of the Japanese Circulation Society)
FRS-076 Correction of Defective Inter-Domain Interactions within Ryanodine Recepter by Dantrolene May be a Potent Therapeutic Strategy against Lethal Arrhythmia(Arrhythmia (basic), The 71st Annual Scientific Meeting of the Japanese Circulation Society)
PJ-402 A Domain Which Has a Strong Effect to Stabilize the Ryanodine Receptor Exists inside the Ryanodine Receptor Itself(Heart failure, basic-5 (M) PJ68,Poster Session (Japanese),The 70th Anniversary Annual Scientific Meeting of the Japanese Circulation
PE-164 Defective Inter-domain Interaction may Cause Spontaneous Ca^ Release via Reduced Affinity of Calmodulin Binding to RyR2 in Failing Hearts(PE028,Heart Failure, Basic (M),Poster Session (English),The 73rd Annual Scientific Meeting of The Japanese
OJ-143 Urinary 8-Hydroxy-2' -Deoxyguanosine Level reflects symptomatic status and severity of systolic dysfunction in Patients with Chronic Heart Failure(Heart failure, clinical(05)(M),Oral Presentation (Japanese),The 72nd Annual Scientific Meeting of the
OJ-243 A-type Natriuretic Peptide Improves Cardiac Function Partly through Suppression of Intracellular ROS in Heart Failure(OJ41,Heart Failure (Treatment) (M),Oral Presentation (Japanese),The 73rd Annual Scientific Meeting of The Japanese Circulation Soc
1 Defective Inter-domain Interaction within the Ryanodine Receptor Underlies a Common Pathogenic Mechanism of Heart Failure and Lethal Arrhythmia(Subcellular Remodeling in the Failing Heart,Symposium 10 (SY-10) (A),The 73rd Annual Scientific Meeting of Th
Dantrolene, a Therapeutic Agent for Malignant Hyperthermia, Inhibits Catecholaminergic Polymorphic Ventricular Tachycardia in a RyR2^ Knock-In Mouse Model
A case of mitochondrial disease with severe left ventricular hypertrophy
Urinary 8-Hydroxy-2'-Deoxyguanosine as a Novel Biomarker for Predicting Cardiac Events and Evaluating the Effectiveness of Carvedilol Treatment in Patients With Chronic Systolic Heart Failure
Low-Dose β-Blocker in Combination With Milrinone Safely Improves Cardiac Function and Eliminates Pulsus Alternans in Patients With Acute Decompensated Heart Failure

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