Fujinaga Koh | Section Of Serology Institute Of Immunological Science Hokkaido University

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著作論文

Dependence of host cell cycle for activation of human immunodeficiency virus type 1 gene expression from latency
Generation of endogenous tumour necrosis factor-α in MOLT-4 cells during the acute replication phase of subsequent latent infection.
Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25+ killer cells in a small subset, leading to induction of apoptosis in bystander cells.
Inhibition of HIV-1 replication by targeting the Rev protein.
A clearer distinction between HIV-1 paired isolates from peripheral blood mononuclear cells of asymptomatic carriers with and without CD8+ T-cells at nef rather than env V3 loci.
Production of doughnut-shaped, protease-defective particles from a T cell clone carrying a provirus with specific mutation in the env, pol, vpr,and nef genes.
E Decoy approach using RNA-DNA chimera oligonucleotides to inhibit the regulatory function of human immunodeficiency virus type 1 Rev protein
Protease-defective, gp120-containing human immunodeficiency virus type 1 particles induce apoptosis more efficiently than does wild-type virus or recombinant gp120 protein in healthy donor-derived peripheral blood T cells.
Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25 killer cells in a small subset, leading to induction of apoptosis in bystander cells
Extracellular Nef protein regulates productive HIV-1 infection from latency.
Nonsense mutations in the vpr gene of HIV-1 during in vitro virus passage and in HIV-1 carrier-derived peripheral blood mononuclear @@S ' cells.
Persistent infection of MT-4 cells by human immunodeficiency virus type 1 becomes increasingly likely with in vitro serial @@S ' passage of wild-type but not nef mutant virus
The carboxyl-terminal region of HIV-1 Nef protein is a cell surface domain that can interact with CD4+ T cells

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