Suzuki Satoko | Section Of Serology Institute Of Immunological Science Hokkaido University

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著作論文

A specific T-cell subset with CD4+/CD38- markers derived from HIV-1 cariers induses apoptosis in healthy donor-derived T-lymphocytes.
Superoxide generation by monocytes following infection with human cytomegalovirus.
Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25+ killer cells in a small subset, leading to induction of apoptosis in bystander cells.
Exposure of Normal Monocyte - Drived Dendritic Cells to Human Immunodeficiency Virus Type - 1 Particles Leads to the Induction of Apoptosis in Co - Cultured CD4^+ as Well as CD8^+ T Cells
Protease-defective, gp120-containing human immunodeficiency virus type 1 particles induce apoptosis more efficiently than does wild-type virus or recombinant gp120 protein in healthy donor-derived peripheral blood T cells.
Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25 killer cells in a small subset, leading to induction of apoptosis in bystander cells

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