Nakaya Takaaki | Institute Of Immunological Science S ' Hokkaido University

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著作論文

DETECTION OF BORNA DISEASE VIRUS RNA IN AUTOPSIED BRAIN SAMPLES OF SCHIZOPHRENIC PATIENTS
Dependence of host cell cycle for activation of human immunodeficiency virus type 1 gene expression from latency
Generation of endogenous tumour necrosis factor-α in MOLT-4 cells during the acute replication phase of subsequent latent infection.
Superoxide generation by monocytes following infection with human cytomegalovirus.
Inhibition of HIV-1 replication by targeting the Rev protein.
Higher prevalence of Borna disease virus infection in blood donors living near thoroughbred horse farms.
A clearer distinction between HIV-1 paired isolates from peripheral blood mononuclear cells of asymptomatic carriers with and without CD8+ T-cells at nef rather than env V3 loci.
High prevalence of Borna disease virus infection in healthy sheep in Japan.
Production of doughnut-shaped, protease-defective particles from a T cell clone carrying a provirus with specific mutation in the env, pol, vpr,and nef genes.
E Decoy approach using RNA-DNA chimera oligonucleotides to inhibit the regulatory function of human immunodeficiency virus type 1 Rev protein
Possible Correlation between Borna Disease Virus Infection and Japanese Patients with Chronic Fatigue Syndrom
Demonstration of Borna disease virus RNA in peripheral blood mononuclear cells from healthy horses in Japan.
Prevalence of Borna disease virus RNA in peripheral blood mononuclear cells from blood donors.
High susceptibility of U937-derived subclone to infection with human immunodeficiency virus type 1 is correlated with virus-induced celldifferentiation and superoxide generation.
Extracellular Nef protein regulates productive HIV-1 infection from latency.
Nonsense mutations in the vpr gene of HIV-1 during in vitro virus passage and in HIV-1 carrier-derived peripheral blood mononuclear @@S ' cells.
Persistent infection of MT-4 cells by human immunodeficiency virus type 1 becomes increasingly likely with in vitro serial @@S ' passage of wild-type but not nef mutant virus
The carboxyl-terminal region of HIV-1 Nef protein is a cell surface domain that can interact with CD4+ T cells
Expression of human immunodeficiency virus type 1 Nef antigen on the

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