IKUTA Kazuyoshi | Hokkaido University

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著作論文

DETECTION OF BORNA DISEASE VIRUS RNA IN AUTOPSIED BRAIN SAMPLES OF SCHIZOPHRENIC PATIENTS
A specific T-cell subset with CD4+/CD38- markers derived from HIV-1 cariers induses apoptosis in healthy donor-derived T-lymphocytes.
Dependence of host cell cycle for activation of human immunodeficiency virus type 1 gene expression from latency
Generation of endogenous tumour necrosis factor-α in MOLT-4 cells during the acute replication phase of subsequent latent infection.
E Detection of Theileria sergenti schizonts in bovine lymph node.
Inhibitory effect of heparin on red blood cell invasion by Theileria sergenti merozoites.
Superoxide generation by monocytes following infection with human cytomegalovirus.
Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25+ killer cells in a small subset, leading to induction of apoptosis in bystander cells.
Inhibition of HIV-1 replication by targeting the Rev protein.
Demonstration of Borna disease virus (BDV) in specific regions of the brain from horses positive for serum antibodies to BDV but negative for BDV RNA in the blood and internal organs.
Higher prevalence of Borna disease virus infection in blood donors living near thoroughbred horse farms.
In vivo dynamics of equine infectious anemia viruses emerging during febrile episodes: Insertions/duplications at the principal neutralizing domain.
A clearer distinction between HIV-1 paired isolates from peripheral blood mononuclear cells of asymptomatic carriers with and without CD8+ T-cells at nef rather than env V3 loci.
A chain section containing epitopes for cytotoxic T, B and helper T cells within a highly conserved region found in the human immunodeficiency virus type 1 Gag protein.
Insertions, duplications and substitutions in restricted gp90 regions of equine infectious anemia virus occurred during febrile episodes in an experimentally infected horse.
Microplate hybridization for Borna disease virus RNA in human peripheral blood mononuclear cells.
High prevalence of Borna disease virus infection in healthy sheep in Japan.
Production of doughnut-shaped, protease-defective particles from a T cell clone carrying a provirus with specific mutation in the env, pol, vpr,and nef genes.
E Decoy approach using RNA-DNA chimera oligonucleotides to inhibit the regulatory function of human immunodeficiency virus type 1 Rev protein
Protease-defective, gp120-containing human immunodeficiency virus type 1 particles induce apoptosis more efficiently than does wild-type virus or recombinant gp120 protein in healthy donor-derived peripheral blood T cells.
Possible Correlation between Borna Disease Virus Infection and Japanese Patients with Chronic Fatigue Syndrom
Demonstration of Borna disease virus RNA in peripheral blood mononuclear cells from healthy horses in Japan.
Prevalence of Borna disease virus RNA in peripheral blood mononuclear cells from blood donors.
Viral activation from latency during retrodifferentiation of U937 cells exposed to phorbol ester followed by infection with human immunodeficiency virus type 1.
High susceptibility of U937-derived subclone to infection with human immunodeficiency virus type 1 is correlated with virus-induced celldifferentiation and superoxide generation.
Stimulation of human immunodeficiency virus type 1 infected cells with superoxide enhances the chemotactic motile response of CD4+ human cells: implication for virus transmission by cell-to-cell interaction.
Amplification of a Full-Length Borna Disease Virus(BDV) cDNA from Total RNA of Cells Persistently Infected with BDV
Maintenance of high virus load even after seroconversion in newborn cats acutely infected with feline immunodeficiency virus.
Superinfection of a defective human immunodeficiency virus type 1 provirus-carrying T cell clone with vif or vpu mutants gives cytopathic virus particles by homologous recombination.
Demonstration of human Borna disease virus RNA in human peripheral blood mononuclear cells.
Naturally occurring accessory gene mutations lead to persistent human immunodeficiency virus type 1 infection of CD4-positive T cells.
Persistent infection of MT-4 cells by human immunodeficiency virus type 1 becomes increasingly likely with in vitro serial @@S ' passage of wild-type but not nef mutant virus
Persistently human immunodeficiency virus type 1-infected T cell CD4 expression.
In vivo induction of human immunodeficiency virus type 1-specific cytotoxic T lymphocytes and delayed-type hypersensitivity by a @@S ' 23-amino acid peptide from the highly conserved region in major @@S ' core protein p24
The carboxyl-terminal region of HIV-1 Nef protein is a cell surface domain that can interact with CD4+ T cells
Multiple effects of CD4 CDR3-related peptide derivatives showing anti-HIV-1 activity on HIV-1 gp120 functions
Superoxide enhances the spread of HIV-1 infection by cell-to-cell
Amplification of superoxide anion generation in phagocytic cells by

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