Kishi Masahiko | Institute Of Immunological Science Hokkaido University

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著作論文

Insertions, duplications and substitutions in restricted gp90 regions of equine infectious anemia virus occurred during febrile episodes in an experimentally infected horse.
Demonstration of Borna disease virus RNA in peripheral blood mononuclear cells from healthy horses in Japan.
Prevalence of Borna disease virus RNA in peripheral blood mononuclear cells from blood donors.
Viral activation from latency during retrodifferentiation of U937 cells exposed to phorbol ester followed by infection with human immunodeficiency virus type 1.
High susceptibility of U937-derived subclone to infection with human immunodeficiency virus type 1 is correlated with virus-induced celldifferentiation and superoxide generation.
Amplification of a Full-Length Borna Disease Virus(BDV) cDNA from Total RNA of Cells Persistently Infected with BDV
Maintenance of high virus load even after seroconversion in newborn cats acutely infected with feline immunodeficiency virus.
Superinfection of a defective human immunodeficiency virus type 1 provirus-carrying T cell clone with vif or vpu mutants gives cytopathic virus particles by homologous recombination.
Demonstration of human Borna disease virus RNA in human peripheral blood mononuclear cells.
Naturally occurring accessory gene mutations lead to persistent human immunodeficiency virus type 1 infection of CD4-positive T cells.
Nonsense mutations in the vpr gene of HIV-1 during in vitro virus passage and in HIV-1 carrier-derived peripheral blood mononuclear @@S ' cells.
Persistent infection of MT-4 cells by human immunodeficiency virus type 1 becomes increasingly likely with in vitro serial @@S ' passage of wild-type but not nef mutant virus
Persistently human immunodeficiency virus type 1-infected T cell CD4 expression.

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