Kameoka Masanori | Institute Of Immunological Science Hokkaido University

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著作論文

Dependence of host cell cycle for activation of human immunodeficiency virus type 1 gene expression from latency
Superoxide generation by monocytes following infection with human cytomegalovirus.
Exposure of resting peripheral blood T cells to HIV-1 particles generates CD25+ killer cells in a small subset, leading to induction of apoptosis in bystander cells.
A clearer distinction between HIV-1 paired isolates from peripheral blood mononuclear cells of asymptomatic carriers with and without CD8+ T-cells at nef rather than env V3 loci.
A chain section containing epitopes for cytotoxic T, B and helper T cells within a highly conserved region found in the human immunodeficiency virus type 1 Gag protein.
Insertions, duplications and substitutions in restricted gp90 regions of equine infectious anemia virus occurred during febrile episodes in an experimentally infected horse.
Production of doughnut-shaped, protease-defective particles from a T cell clone carrying a provirus with specific mutation in the env, pol, vpr,and nef genes.
Protease-defective, gp120-containing human immunodeficiency virus type 1 particles induce apoptosis more efficiently than does wild-type virus or recombinant gp120 protein in healthy donor-derived peripheral blood T cells.
Viral activation from latency during retrodifferentiation of U937 cells exposed to phorbol ester followed by infection with human immunodeficiency virus type 1.
High susceptibility of U937-derived subclone to infection with human immunodeficiency virus type 1 is correlated with virus-induced celldifferentiation and superoxide generation.
Stimulation of human immunodeficiency virus type 1 infected cells with superoxide enhances the chemotactic motile response of CD4+ human cells: implication for virus transmission by cell-to-cell interaction.
Extracellular Nef protein regulates productive HIV-1 infection from latency.
Cytotoxic T lymphocyte response in mice induced by a recombinant BCG Vaccination which produces an extracellular α antigen that @@S ' fused with the human immunodeficiency virus type 1 envelope immuno @@S dominant domain in the V3 loop.
In vivo induction of human immunodeficiency virus type 1-specific cytotoxic T lymphocytes and delayed-type hypersensitivity by a @@S ' 23-amino acid peptide from the highly conserved region in major @@S ' core protein p24
The carboxyl-terminal region of HIV-1 Nef protein is a cell surface domain that can interact with CD4+ T cells
Superoxide enhances the spread of HIV-1 infection by cell-to-cell
Poly(ADP-ribose) polymerase activity in various U937 cell subclones with different susceptibility to HIV-1 infection : Its dramatic decre ase following persistent virus infection.
AIDS pathogenesis: the role of accessory gene mutations, leading to formation of long-lived persistently infected cells and/or apoptosis- inducing HIV-1 particles.

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