INHIBITION OF THE POSITIVE INOTROPIC EFFECT OF DOPAMINE BY YM-09538 WITH α- AND β-ADRENOCEPTOR ANTAGONISTIC ACTION IN THE RABBIT PAPILLARY MUSCLE
スポンサーリンク
概要
著者
-
Endoh Masao
Department of Cardiovascular Pharmacology, Yamagata University School of Medicine
-
Endoh Masao
Department Of Cardiovascular Pharmacology Yamagata University School Of Medicine
-
ENDOH Masao
Department of Pharmacology, Tohoku University School of Medicine
関連論文
- Receptor Subtypes Mediating the Inotropic Effects and Ca2+ Signaling Induced by Endothelin-1 Through Crosstalk With Norepinephrine in Canine Ventricular Myocardium
- Cardiac-specific Overexpression of Diacylglycerol Kinase ζ Prevents Angiotensin II-induced Cardiac Hypertrophy in Transgenic Mice(Cardiac Hypertrophy 1 (M), The 69th Annual Scientific Meeting of the Japanese Circulation Society)
- Cardiac Ca^ Signaling and Ca^ Sensitizers
- The G-Protein G_0 in Mammalian Cardiac Muscle: Localization and Coupling to A_1 Adenosine Receptors
- The State of Cardiac Contractile Proteins during the Diastolic Phase : SYMPOSIUM ON HEART MECHANICS IN DIASTOLE
- Receptor Subtypes Mediating the Inotropic Effects and Ca^ Signaling Induced by Endothelin-1 Through Crosstalk With Norepinephrine in Canine Ventricular Myocardium
- Wortmannin Inhibits the Increase in Myofilament Ca^ Sensitivity Induced by Cross-Talk of Endothelin-1 With Norepinephrine in Canine Ventricular Myocardium
- Differential Inotropic Effects of Endothelin-1, Angiotensin II, and Phenylephrine Induced by Crosstalk With cAMP-Mediated Signaling Process in Dog Ventricular Myocardium
- FAILURE OF DIBUTYRYL AND 8-BROMO-CYCLIC GMP TO MIMIC THE ANTAGONISTIC ACTION OF CARBACHOL ON THE POSITIVE INOTROPIC EFFECTS OF SYMPATHOMIMETIC AMINES IN THE CANINE ISOLATED VENTRICULAR MYOCARDIUM
- EFFECT OF MANGANESE IONS ON THE CONTRACTION AND AUTOMATICITY OF THE BLOOD-PERFUSED CANINE PAPILLARY MUSCLE
- INHIBITION OF THE POSITIVE INOTROPIC EFFECT OF DOPAMINE BY YM-09538 WITH α- AND β-ADRENOCEPTOR ANTAGONISTIC ACTION IN THE RABBIT PAPILLARY MUSCLE
- ADRENOCEPTORS MEDIATING THE POSITIVE INOTROPIC EFFECT IN THE CANINE ATRIAL MYOCARDIUM
- CORRELATION OF CYCLIC AMP AND CYCLIC GMP LEVELS WITH CHANGES IN CONTRACTILE FORCE OF DOG VENTRICULAR MYOCARDIUM DURING CHOLINERGIC ANTAGONISM OF POSITIVE INOTROPIC ACTIONS OF HISTAMINE, GLUCAGON, THEOPHYLLINE AND PAPAVERINE
- Pharmacological Characteristics of Inhibitory Action of the Selective α_1-Antagonist JTH-601 on the Positive Inotropic Effect Mediated by α_1-Adrenoceptors in Isolated Rabbit Papillary Muscle
- INTRACELLULAR CALCIUM MOBILIZATION AND ITS MODULATION BY PHYSIOLOGICAL AND PATHOPHYSIOLOGICAL INTERVENTIONS IN AEQUORIN-INJECTED MYOCARDIAL CELLS
- Physiological and Pathophysiological Modulation of Calcium Signaling in Myocardial Cells : 54th Annual Scientific Session of the Japanese Circulation Society
- SUBCELLULAR MECHANISMS UNDERLYING THE INHIBITORY ACTION OF MUSCARINIC CHOLINERGIC RECEPTOR AGONISTS ON MAMMALIAN MYOCARDIAL CONTRACTILITY IN RELATION TO CHANGES IN INTRACELLULAR CALCIUM TRANSIENTS
- (-)-Enantiomer EMD 57439 Antagonizes the Ca^ Sensitizing Effect of (+)-Enantiomer EMD 57033 on Diastolic Function but Not on Systolic Function in Rabbit Ventricular Cardiomyocytes
- Differential effects of isoproterenol on the canine atrial action potential in the presence of carbachol or nicorandil.
- Involvement of cyclic AMP in the positive inotropic effect of OPC-8212, a new cardiotonic agent, on the canine ventricular muscle.
- Effects of a new cardiotonic agent 1,2-dihydro-6-methyl-2-oxo-5-(imidazo(1,2-a)pyridin-6-yl)-3-pyridine carbonitrile hydrochloride monohydrate (E-1020) on contractile force and cyclic AMP metabolism in canine ventricular muscle.
- Inotropic Effects of Staurosporine, NA 0345 and H-7, Protein Kinase C Inhibitors, on Rabbit Ventricular Myocardium: Selective Inhibition of the Positive Inotropic Effect Mediated by .ALPHA.1-Adrenoceptors.
- The Na+-Ca2+ Exchanger Contributes to .BETA.-adrenoceptor Mediated Positive Inotropy in Mouse Heart.
- Changes in Intracellular Ca2+ Mobilization and Ca2+ Sensitization as Mechanisms of Action of Physiological Interventions and Inotropic Agents in Intact Myocardial Cells.