FRS-031 Midkine Prevents Cardiac Remodeling in Rabbit Congestive Heart Failure through Its Anti-apoptotic Effect(FRS7,Challenging Approaches to Heart Failure Treatment (M),Featured Research Session (English),The 73rd Annual Scientific Meeting of The Japan
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概要
- 論文の詳細を見る
- 社団法人日本循環器学会の論文
- 2009-03-01
著者
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児玉 逸雄
名古屋大学環境医学研究所循環器部門
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児玉 逸雄
名古屋市立東市民病院
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児玉 逸雄
静岡済生会総合病院 循環器内科
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Kodama Itsuo
Department of Cardiovascular Research, Research Institute of Environmental Medicine, Nagoya Universi
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兜玉 逸雄
名古屋大学環境医学研究所 心・血管分野
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Kodama Itsuo
静岡済生会総合病院 不整脈科
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Kodama Itsuo
J-rhythm Study
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HARADA Masahide
Department of Energy Conversion Engineering, Kyushu University
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KAMIYA Kaichiro
Department of Humoral Regulation Division of Regulation of Organ Function Research Institute of Envi
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Kodama Itsuo
山口大学 医学系研究科器官病態内科学循環器内科
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Kodama Itsuo
東京医科歯科大学生体材料工学研究所
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Kodama Itsuo
山口大学 循環器内科
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児玉 逸雄
名古屋市総合リハビリテーションセンター 企画研究局
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Kodama Itsuo
名古屋大学環境医学研究所
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Honjou Haruo
Department Of Humoral Regulation Research Institute Of Environmental Medicine Nagoya University
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Kamiya Kaichiro
静岡済生会総合病院 不整脈科
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Kamiya K
Division Of Cardiovascular Research Reseearch Institute Of Environmental Medicine Nagoya University
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Kamiya Kaichiro
名古屋大学環境医学研究所 循環器学
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Kamiya K
Nagoya Univ. Nagoya Jpn
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Kamiya Kaichiro
Department Of Humoral Regulation The Research Institute Of Environmental Medicine Nagoya University
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Mori H
Department Of Bioscience Department Of Cardiovascular Surgery
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Kodama Itsuo
Department Of Circulation Division Of Regulation Of Organ Function Research Institute Of Environment
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Horiba Mitsuru
Department of Bio-information Analysis, Research Institute of Environmental Medicine
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Tsuji Yukiomi
Department of Cardiovascular Research, Research Institute of Environmental Medichine, Nagoya Univers
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Tsuji Y
Department Of Cardiovascular Research Research Institute Of Environmentl Medicine Nagoya University
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Tsuji Yukiomi
名古屋大学環境医学研究所 循環器学
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Tsuji Y
Nagoya Univ. Nagoya Jpn
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Tsuji Yukiomi
Department Of Cardiology Nagoya University Graduate School Of Medicine
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Horiba Mitsuru
名古屋大学環境医学研究所 循環器学
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Sumida Arihiro
Department of Cardiovascular Research, Research Institute of Environmental Medicine, Nagoya Universi
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Harada Masahide
Division Of Arrhythmia And Electrophysiology Shizuoka Saiseikai General Hospital
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Hojo Mayumi
Department of Cardiovascular Research, Research Institute of Environmental Medicine (RIEM), Nagoya U
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Sumida Arihiro
Department Of Cardiovascular Research Research Institute Of Environmentl Medicine Nagoya University
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Sumida Arihiro
Department Of Cardiology Japanese Red Cross Nagoya 1st Hospital
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児玉 逸雄
名古屋大学環境医学研究所
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Kamiya Kaichiro
Department Of Cardiovascular Research Research Institute Of Environmental Medicine Nagoya University
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Kamiya Kaichiro
Division Of Circulation And Respiration Research Institute Of Environmental Medicine Nagoya Universi
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Mitamura Hideo
Division Of Cardiology Tokyo Saiseikai Central Hospital
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Horie Minoru
Department Of Cardiopulmonary Medicine Shiga University Of Medical Science
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Harada Masahide
Department Of Cardiology Tokyo Medical University
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Harada Masahide
Department Of Circulation Research Institute Of Environmental Medicine
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Hone Minoru
Department Of Cardiovascular And Respiratory Medicine Siga University Of Medical Science
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Kodama Itsuo
Department Of Cardiovascular Research R.i.e.m. Nagoya University
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Hojho Mayumi
Department Of Circulation Research Institute Of Environmental Medicine
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Harada Masahide
Department Of Cardiovascular Research Research Institute Of Environmental Medicine Nagoya University
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Kamiya K
Nagoyauniv. Nagoya Jpn
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Tsuji Yukiomi
Cardiovascular Center Nagoya Dai-ni Red Cross Hospital
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Hojo Mayumi
Department Of Cardiovascular Research Research Institute Of Environmental Medicine (riem) Nagoya Uni
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本荘 晴朗
Department Of Cardiovascular Research Research Institute Of Environmental Medicine Nagoya University
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Kamiya kaichiro
Department of Cardiovascular Research, Research Institute of Environmental Medicine, Nagoya University
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- Altered expression of connexin43 contributes to the arrhythmogenic substrate during the development of heart failure in cardiomyopathic hamster
- Heavy ion radiation up-regulates Cx43 and ameliorates arrhythmogenic substrates in hearts after myocardial infarction
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