尿中線溶活性の研究 : 第三報高圧酸素療法と血中および尿中線溶活性の動態について

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Exposure of subjects to oxygen at increased pressure has lately been found of several clinical applications. Hyperbaric oxygen has been ad vocated in the management of certain medical and surgical conditions. However, the effect of oxygen at high atmospheric pressure (OHP) on the living body, especially on fibrinolytic activity remains unknown. This study was undertaken to determine, in 20 cases of urological diseases with hematuria, the effect of oxygen at 3 atmospheres absolute (3 ATA) on the fibrinolytic activities of circulating blood and urine. Whole plasmin activity was found to be increased but activities of proactivator, Plasminogen activator and antiplasmin were not found to be changed. These changes are believed to be caused by stress brought about by oxygen at increased tensions. On the other hand, oxygen at 3 atmosphere pressure produced an decrease in the fibrinolytic activities of urine (fresh urine and extract of urine prepared with 2M-KSCN). It is suggested that this change may in part be caused by renal vasoconstriction brought about by oxygen at increased tensions and by oxygen poisoning of the renal tubular cell enzyme systems. These seem to indicate that the kidneys act not as regulators of circulating fibrinolytic activity but as independent contributors to this actiyity and that the quantities provided-so far as at present is known-are determined by the rate of blood flow through the kidneys.
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尿中線溶活性の研究 : 第三報高圧酸素療法と血中および尿中線溶活性の動態について

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尿中線溶活性の研究 : 第三報 高圧酸素療法と血中および尿中線溶活性の動態について

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尿中線溶活性の研究 : 第三報高圧酸素療法と血中および尿中線溶活性の動態について