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Department Of Pharmacology Sapporo Medical College | 論文
- EVIDENCE FOR CENTRAL ALPHA_2-ADRENERGIC MECHANISM OF CLONIDINE-INDUCED EJACULATORY DISTURBANCE IN DOGS
- Ion channels and diseases
- POTENTIATION OF TOLUENE TOXICITY BY HEPATIC ENZYME INHIBITION IN MICE
- HEPATIC RESPONSES TO ADRENERGIC AND CHOLINERGIC STIMULATION IN CCl_4-TREATED RATS
- EFFECTS OF ETHANOL ON THE G PROTEIN AND TUBULIN THAT REGULATE NEURONAL SIGNAL TRANSDUCTION
- POTENTIATION OF ISOPROTERENOL-INDUCED SALIVATION BY LABETALOL
- A POSSIBLE ROLE OF ALPHA ADRENERGIC RECEPTORS IN SALIVATION INDUCED BY ISOPROTERENOL IN MICE
- STIMULATING EFFECT OF α-ADRENOCEPTOR AGONISTS ON ISOPROTERENOL-INDUCED AMYLASE RELEASE IN RAT PAROTID TISSUE
- INCREASE IN POTASSIUM RELEASE FROM RAT PAROTID TISSUE REPEATEDLY EXPOSED TO ISOPROTERENOL
- Relationship between glucagon-induced cyclic AMP accumulation and DNA synthesis in carbon tetrachloride-intoxicated rat liver.
- .BETA.1-Adrenoceptor-mediated amylase release and cyclic AMP accumulation in rat parotid gland tissue.
- Increase of .ALPHA.1-adrenoceptor binding after exposure to isoproterenol in rat parotid glands.
- .BETA.1-Adrenergic regulation of cyclic nucleotide levels and potassium fluxes in rat parotid gland.
- Isoproterenol inhibition of potassium release from rat parotid gland.
- Effects of repeated exposure to isoproterenol in vivo and in vitro on amylase release from rat parotid tissue.
- Alteration in Ca2+/Calmodulin-sensitive Adenylyl Cyclase Activity in the Hippocampus and the Cerebral Cortex of Stroke-prone Spontaneously Hypertensive Rats