Komatsu Kenshi | Dept. Genome Repair Dynamics Rad. Biol. Center Kyoto Univ.

スポンサーリンク

概要

関連著者

もっと見る

著作論文

98 Localization of Rad51 protein in cells from patients with Fanconi Anemia D1 group(Repair of radiation damage, Abstracts of the 46th Annual Meeting of the Japan Radiation Research Society)
p53-Binding Protein 1 (53BP1) is Involved in the Repair of DNA Double-Strand Breaks
Histone H2AX regulates the formation of NBS1 foci on DNA damage sites
Dissociation of ATM/NBS1 functions in telomere maintenance
Bystander mutagenic effect of alpha particle in human-hamster hybrid cells
Somatic Mutation Caused by Low Dose Rate Tritium Radiation : Studies Using A Hyper-sensitive DetectionSystem
Complementation studies of immortalized cell lines from infants with total PCS
Establishement of mouse Nbs1-deficient cell lines by gene targeting
Association of cells from Fanconi anemia patients with radiation sensitivity
Association of DNA repair protein NBS1 with telomere maintenance
The function of NBS1 and histone H2AX in the early process of DNA repair.
Reversed dose-rate effect of high LET radiation in mutation induction
Chromosomal instability syndrome of total premature chromatid separation with mosaic variegated aneuploidy is defective in mitotic spindle checkpoint
Screening of protein interacting with FANCG by Yeast two-hybrid system
Functionally important domain analysis of the Nijimegen breakage syndrome gene. NBS1
Development of a Radio-sensitive Mutation System for the Study of Dose Rate Effects of Space High LET Radiation
NBS1, the Nijmegen breakage syndrome protein, regulates the localization of DNA repair complex hRAD50/hMRE11/NBS1
Accelerated telomere shortening by disruption of DNA repair protein NBS1
Construction of Nbs1 knockout cell line using chicken DT40
Nijmegen Breakage Syndrome (NBS) gene, NBS1, is involved in telomeric length maintenance.
Loss of p53 -dependent signal transdution in γ -ray irradiated scid mouse fibroblasts
Positional cloning of the gene for Nijmegen breakage syndrome
Genetic Mapping of Nijmegen Breakage Syndrome using Functional Complementation Assays and Homozygosity Mapping
Haplotype Analysis of Nijmegen Breakage Syndrome and Construction of Physical Map of Candidate Region.
Complementation Assay between Radiation-Sensitive irs2 Mutant and Ataxia-Telangiectasia Cell
The Genetic Homogeneity of Nijmegen Breakage Syndrome V1 and V2.
Mapping of the underlying gene for rare genetic complementation group Fanconi anemia
NBS1 Regulates Apoptosis Induced by γ-Irradiation in Lymphoblastoid
Mutation screening of the NBS1 gene in sporadic malignant lymphoma
LET, cell cycle dependence for mutation induction and spectrum in HPRT locus
Radiation-induced internal developmental abnormalities in scid mice
Mutation Analysis of the Fanconi Anemia Gene FAA in Japanese patients
Mutation Analysis of the Fanconi Anemia Genes in Japanese Patients
Function of NBS1 in Activation of ATM kinase in Response to IR.
Heat sensitivity of DNA-dependent protein kinase activity
The genetic complementation assay of Nijmegen breakage syndrome witit ataxia telangiectasia on the basis of radiation sensitivity to cell killing.
Nijmegen breakage syndrome : Sequence analysis of an 800-kb genomic region on chromosome 8q21.
The scid factor on human chromosome 8 restores both V(D)J recombination and double-strand break repair
The relationship between NBS1 and cell death induced by γ-ray irradiation in lymphoblastoid
Cell cycle and LET dependence for 6-thioguanine-resistant mutation induced by heavy ion beam in mouse L5178Y cells
Recovery of heat inactivated DNA-PK activity and HSC73
Recovery of heat inactivated DNA-PK activity and repair of DNA double strand break
Genetic heterogeneity of ataxia-telangiectasia-like hamster mutant irs2 with Nijmegen breakage syndrome
Genetic heterogeneity of ataxia-telangiectasia-like hamster mutant irs2 with Nijmegen breakage syndrome.
Radiation-induced HPRT mutation of a radiation sensitive hamster cell line irs1SF
DNA-dependent protein kinase restores mouse scid mutation
Inhibition of radiation-induced oncogenic transformation in C3H10T1/2 cells by green tea component, (-)-epigallocatechin gal late.

もっと見る

スポンサーリンク