Yoneda Yukio | Laboratory Of Molecular Pharmacology Division Of Pharmaceutical Sciences Kanazawa University Graduat

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著作論文

Inhibition by 2-Methoxy-4-ethylphenol of Ca2+ Influx Through Acquired and Native N-Methyl-D-aspartate–Receptor Channels
Predominant Promotion by Tacrolimus of Chondrogenic Differentiation to Proliferating Chondrocytes
Neurogenesis Mediated by γ-Aminobutyric Acid and Glutamate Signaling
Gradual Downregulation of Protein Expression of the Partner GABABR2 Subunit During Postnatal Brain Development in Mice Defective of GABABR1 Subunit
Neonatal Phencyclidine Treatment in Mice Induces Behavioral, Histological and Neurochemical Abnormalities in Adulthood
Functional Proteins Involved in Regulation of Intracellular Ca^ for Drug Development : Preface
Increased GABA Transport Activity in Rat Calvarial Osteoblasts Cultured under Hyperglycemic Conditions(Pharmacology)
nrf2 Expressed by Bone(International Symposium of Maxillofacial and Oral Regenerative Biology in Okayama 2005)
Release of Endogenous Glutamate by AMPA Receptors Expressed in Cultured Rat Costal Chondrocytes(Pharmacology)
Glutamate Signaling System in Bone
Pharmacological Topics of Bone Metabolism : Glutamate as a Signal Mediator in Bone
Functional Proteins Involved in Regulation of Intracellular Ca^ for Drug Development : Desensitization of N-Methyl-D-aspartate Receptor Channels
Transcriptional Regulation of Neuronal Genes and Its Effect on Neural Functions : Gene Expression in Response to Static Magnetism in Cultured Rat Hippocampal Neurons
Neurogenesis Mediated by GABA and Glutamate Signaling
Ifenprodil severely retards P19 neuronal differentiation via polyamine site of NR2B subunit on NMDA receptors
A Negative Correlation Between Expression Profiles of Runt-Related Transcription Factor-2 and Cystine/Glutamate Antiporter xCT Subunit in Ovariectomized Mouse Bone
A Tale of Early Response Genes
Functional Proteins Involved in Regulation of Intracellular Ca2+ for Drug Development: Desensitization of N-Methyl-D-aspartate Receptor Channels
Selective Upregulation of Per1 mRNA Expression by ATP Through Activation of P2X7 Purinergic Receptors Expressed in Microglial Cells
Possible Involvement of Glutamatergic Signaling Machineries in Pathophysiology of Rheumatoid Arthritis
Inhibition by 2-Methoxy-4-ethylphenol of Ca^ Influx Through Acquired and Native N-Methyl-D-aspartate-Receptor Channels
Selective Downregulation of N-methyl-D-aspartate Receptor (NMDAR) rather than Non-NMDAR Subunits in Ipsilateral Cerebral Hemispheres in Rats with Middle Cerebral Artery Occlusion
Gradual Downregulation of Protein Expression of the Partner GABA_BR2 Subunit During Postnatal Brain Development in Mice Defective of GABA_BR1 Subunit
Possible Involvement of Glutamatergic Signaling Machineries in Pathophysiology of Rheumatoid Arthritis
Selective Upregulation of Per1 mRNA Expression by ATP Through Activation of P2X7 Purinergic Receptors Expressed in Microglial Cells
A Negative Correlation Between Expression Profiles of Runt-Related Transcription Factor-2 and Cystine/Glutamate Antiporter xCT Subunit in Ovariectomized Mouse Bone
Amelioration by the Natural Polyamine Spermine of Cartilage and Bone Destruction in Rats With Collagen-Induced Arthritis
Delayed Mitochondrial Membrane Potential Disruption by ATP in Cultured Rat Hippocampal Neurons Exposed to N-Methyl-D-Aspartate
Possible Modulation of Process Extension by N-Methyl-D-aspartate Receptor Expressed in Osteocytic MLO-Y4 Cells
In Vivo and In Vitro Treatment With Edaravone Promotes Proliferation of Neural Progenitor Cells Generated Following Neuronal Loss in the Mouse Dentate Gyrus
A Negative Correlation Between Per1 and Sox6 Expression During Chondrogenic Differentiation in Pre-chondrocytic ATDC5 Cells
Prevention of Bone Loss after Ovariectomy in Mice with Preferential Overexpression of the Transcription Factor Paired Box-5 in Osteoblasts
In Vivo and In Vitro Treatment With Edaravone Promotes Proliferation of Neural Progenitor Cells Generated Following Neuronal Loss in the Mouse Dentate Gyrus
A Negative Correlation Between Per1 and Sox6 Expression During Chondrogenic Differentiation in Pre-chondrocytic ATDC5 Cells

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